in hcm patients, myocardial fibrosis can cause the late diastolic capacity recovery time delay, but no significant difference between the filling rate with no fibrosis.

conclusion: avp stimulates collagen synthesis and no synthesis in neonatal rat cfs, no contents enhancement could inhibit cfs collagen synthesis and myocardial fibrosis induced by avp.

结论：AVP促进CFs胶原合成和NO合成，NO合成增加能够拮抗AVP的促CFs胶原合成作用，延缓心肌纤维化发生。

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conclusions serum level of p ⅲ p is a useful marker reflecting the degree of left ventricular remodeling after ami. collagen response and myocardium fibrosis are reduced after reperfusion of the infarct related artery and left ventricular function preserved.

结论AMI后血清PⅢP浓度的变化是反映左室重构有用的指标，IRA再灌注可以明显减少梗死后的胶原形成，防止心肌纤维化的形成，改善左室功能。

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in order to comprehend better pathological mechanisms of myocardial fibrosis and provide bases for clinical prevention and therapy, the pathomorphological and quantitive changes of the collagen types i and iii were investigated in this study.

本研究通过观察上述心肌纤维化间质I、III型胶原的病理形态改变并定量分析，为深入了解心肌纤维化的病理机制以及临床防治提供依据；

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experimental research on pathogenesis of cardiac fibrosis and effects of felodipine on metabolic syndrome; analysis of causes of relapse chest pain in early stage after percutaneous coronary intervention for acute coronary syndrome

代谢综合征心肌纤维化发病机制及药物干预的研究急性冠脉综合征PCI术后近期再发胸痛原因分析

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conclusion angiotension converting enzyme inhibitor combined with simvastatin is helpful to reduce the myocardial fibrosis and to improve the left ventricular hypertrophy and diastolic function in eh patients.

结论：EH患者在接受血管紧张素转换酶抑制药同时，再联合使用辛化他汀，能明显减轻心肌纤维化程度，改善左室肥厚和舒张功能。

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there are no reports about the effect and mechanism of bmp-7 on myocardial fibrosis after mi.

目前国内外尚无BMP-7对心梗后心肌纤维化的影响及其作用机制的研究报道。

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in previous animal experiments, we have found that tp could inhibit myocardial fibrosis of the spontaneously hypertensive rat, but the exact mechanism was not yet clarified.

本课题组之前的动物实验研究发现，TP能够抑制自发性高血压大鼠的心肌纤维化，但具体机制尚未阐明。

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conclusions elevation of extracellular matrix turnover can be monitored in the serum by determining the circulation levels of p ⅲ p and ln associated with myocardial fibrosis and can predict the clinical stage of heart failure

结论PⅢP及LN水平的升高可能部分与心肌纤维化有关，可在充血性心力衰竭患者的血清中检测出来，并能反映心衰的严重程度

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study of the relationship between cardiac fibrosis and macrophages in spontaneously hypertensive rats ( shrs)-the interventional experiment of benazepril

自发性高血压大鼠（SHR）心肌纤维化和巨噬细胞关系的研究&苯那普利干预试验

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the area of myocardial fibrosis was measured by imaging analysis system;

图像分析系统检测心肌纤维化面积，计算胶原容积分数（CVF）；

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perindopril can inhibit progress of rh rat myocardial fibrosis by inhibiting the expression of mmp-2 and tgf-β 1.

培哚普利可通过降低TGF-β1和MMP-2的表达进而抑制肾性高血压大鼠心肌纤维化的进展。

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acei has a regulating role for cytokine levels and myocardial fibrosis in patients with chf.

ACEI对CHF患者促炎性细胞因子和心肌纤维化水平具有调控作用。

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the effect of early los treatment on blood pressure and myocardial fibrosis in spontaneously hypertensive rats

早期氯沙坦治疗对SHR大鼠血压和心肌纤维化的影响

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objective: to explore the effects of aldosterone antagonism spironolactone on cardiac fibrosis in patients with essential hypertension.

目的：观察醛固酮拮抗剂&螺内酯对原发性高血压（EH）患者心肌纤维化的影响。

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methods myocardial fibrosis model of rats was built by injection of iso for 7 days [ 5 mg/ ( kg · d), sc].

方法用Iso5mg/（kg·d）背部皮下注射，连续7天，建立大鼠心肌纤维化模型。

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the pretreatment of spc followed by cp can prevent and relieve the development of myofibrosis and cga.

SPC和CP联合预处理，可以预防和减缓移植心脏心肌纤维化和心脏动脉硬化。

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supplementation se and/ of ve to the diet could alleviate these changes in different levels, lessen and reverse the myocardial fibrosis and myocardial remodel.

膳食中补充硒和/或维生素E可以不同程度的缓解这些改变，改善心肌纤维化和心肌重构。

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conclusions acoustic densitometry technology is feasible and sensitive in detecting and monitoring the degree of left atrial myocardial fibrosis in elder essential hypertensive patients.

结论AD技术是检测老年人高血压左房心肌纤维化程度敏感而可行的方法。

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their receptor antagonists may be useful in preventing cardiac fibrosis and remolding.

应用ANGⅡ和ALD的受体拮抗剂可抑制心肌纤维化，预防心室重塑的发生。

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conclusions: tranilast can prevent left ventricular fibrosis and improve left ventricular function after myocardial infarction.

结论：曲尼司特可减轻AMI后左室心肌纤维化，改善左室功能。

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