conclusion angiotension converting enzyme inhibitor combined with simvastatin is helpful to reduce the myocardial fibrosis and to improve the left ventricular hypertrophy and diastolic function in eh patients.

  • 结论:EH患者在接受血管紧张素转换酶抑制药同时,再联合使用辛化他汀,能明显减轻心肌纤维化程度,改善左室肥厚和舒张功能。
  • 来源:互联网摘选更新时间:2026-07-13 12:29:14

  • 重点词汇
  • convertingn.吹(炉冶)炼,转炉炼钢;
  • theart.这个;指已提到或易领会到的人或事物;指独一无二的、正常的或不言而喻的人或事物;用以泛指;与形容词连用,指事物或统称的人;用于姓氏的复数形式前,指家庭或夫妇;(指特定用途的事物)足够,恰好;每,一;当前的,本,此;(重读,表示所指的为知名或重要的人或事物)
  • inhibitorn.抑制剂,抑制者;抗老化剂;
  • diastolicadj.心脏舒张的;
  • ventricularadj.心室的;(心)室的
  • simvastatin[医]系HMG辅酶A还原酶抑制剂,能降低男性血浆胆固醇和减少胆固醇在动脉沉积;
  • simvastatin怎么记n.Neovastatin; Simvastatin;
  • myocardialadj.心肌的;
  • 相关例句
1、

there are no reports about the effect and mechanism of bmp-7 on myocardial fibrosis after mi.

目前国内外尚无BMP-7对心梗后心肌纤维化的影响及其作用机制的研究报道。

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2、

in previous animal experiments, we have found that tp could inhibit myocardial fibrosis of the spontaneously hypertensive rat, but the exact mechanism was not yet clarified.

本课题组之前的动物实验研究发现,TP能够抑制自发性高血压大鼠的心肌纤维化,但具体机制尚未阐明。

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3、

conclusions elevation of extracellular matrix turnover can be monitored in the serum by determining the circulation levels of p ⅲ p and ln associated with myocardial fibrosis and can predict the clinical stage of heart failure

结论PⅢP及LN水平的升高可能部分与心肌纤维化有关,可在充血性心力衰竭患者的血清中检测出来,并能反映心衰的严重程度

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4、

study of the relationship between cardiac fibrosis and macrophages in spontaneously hypertensive rats ( shrs)-the interventional experiment of benazepril

自发性高血压大鼠(SHR)心肌纤维化和巨噬细胞关系的研究&苯那普利干预试验

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5、

the area of myocardial fibrosis was measured by imaging analysis system;

图像分析系统检测心肌纤维化面积,计算胶原容积分数(CVF);

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6、

perindopril can inhibit progress of rh rat myocardial fibrosis by inhibiting the expression of mmp-2 and tgf-β 1.

培哚普利可通过降低TGF-β1和MMP-2的表达进而抑制肾性高血压大鼠心肌纤维化的进展。

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7、

acei has a regulating role for cytokine levels and myocardial fibrosis in patients with chf.

ACEI对CHF患者促炎性细胞因子和心肌纤维化水平具有调控作用。

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8、

the effect of early los treatment on blood pressure and myocardial fibrosis in spontaneously hypertensive rats

早期氯沙坦治疗对SHR大鼠血压和心肌纤维化的影响

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9、

objective: to explore the effects of aldosterone antagonism spironolactone on cardiac fibrosis in patients with essential hypertension.

目的:观察醛固酮拮抗剂&螺内酯对原发性高血压(EH)患者心肌纤维化的影响。

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10、

methods myocardial fibrosis model of rats was built by injection of iso for 7 days [ 5 mg/ ( kg · d), sc].

方法用Iso5mg/(kg·d)背部皮下注射,连续7天,建立大鼠心肌纤维化模型。

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11、

the pretreatment of spc followed by cp can prevent and relieve the development of myofibrosis and cga.

SPC和CP联合预处理,可以预防和减缓移植心脏心肌纤维化和心脏动脉硬化。

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12、

supplementation se and/ of ve to the diet could alleviate these changes in different levels, lessen and reverse the myocardial fibrosis and myocardial remodel.

膳食中补充硒和/或维生素E可以不同程度的缓解这些改变,改善心肌纤维化和心肌重构。

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14、

their receptor antagonists may be useful in preventing cardiac fibrosis and remolding.

应用ANGⅡ和ALD的受体拮抗剂可抑制心肌纤维化,预防心室重塑的发生。

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15、

conclusions: tranilast can prevent left ventricular fibrosis and improve left ventricular function after myocardial infarction.

结论:曲尼司特可减轻AMI后左室心肌纤维化,改善左室功能。

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16、

conclusion dsp and combined use of dsp and fosinopril can decrease left ventricular fibrosis.

结论DSP与福辛普利联用可改善SHR心肌纤维化(MF)。

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17、

backgrounds: myocardial fibrosis refers to disproportionate increase of myocardial collagen fibers in a variety of factors.

背景:心肌纤维化(myocardial fibrosis,MF)是指在各种致纤维化因素的作用下心肌胶原纤维不成比例的增多。

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18、

the control group was the model rats received only dmem injection.

方法:大鼠经皮下注射Iso共10天以制备心肌纤维化模型,心肌线粒体以差速离心法分离。对照组仅注射无血清的培养液。

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19、

the effect of targeting inhibition of connective tissue growth factor on myocardial fibrosis by rna interference

RNA干扰靶向抑制结缔组织生长因子对心肌纤维化的影响

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20、

nf-κ b contributes to the formation of myocardial fibrosis.

NF-κB参与了心肌纤维化的形成。

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