pre-synaptic

The adenosine exerts its inhibitory effect through activation of both pre-synaptic and post-synaptic adenosine A1 receptors ( A1R's). Thus, A1 receptors play an important role in promoting sleep.

腺苷是通过激活突触前膜和突触后膜腺苷A1受体而起抑制性作用，所以腺苷A1受体在促进睡眠中起重要作用。

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Pre synaptic long-term potentiation of synaptic transmission in the pathway from auditory cortex to medial geniculate body

听皮层-内侧膝状体突触传递的长时程增强及其突触前机制的研究

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CCK-8S is one of the strongest endogenous anti-opioid substances and suppresses opioid peptides-mediated 'pre-synaptic inhibition' of γ-aminobutyric acid ( GABA) release.

CCK-8S是目前发现最强的内源性抗阿片物质。阿片肽可增强γ-氨基丁酸（GABA）介导的突触前抑制的作用，而CCK-8S可对抗阿片肽的这一作用。

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DR increases the expression of SYN of cortical and hippocampal neurons by improving the injury of cholinergic neuron to recovery the function of synaptic transmission.

地黄饮子能通过改善鼠脑神经元胆碱能损害，来上调海马和皮层神经元SYN蛋白的表达，使突触传递功能恢复。

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Pre-synaptic test stimulation was given at 0.07 Hz intervals through bipolar stimulating electrodes. EPSCs of layer ⅱ~ ⅳ were recorded by recording electrodes filled with 0.5% biocytin.

通过视皮层的双极刺激电极给予0.07Hz的突触前测试刺激，记录视皮层神经元的PSCs。

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Objective To further study the pathological characteristics at pre and post synaptic membranes of the neuromuscular junction ( NMJ) in patients with myasthenia gravis ( MG) and their significance.

目的探讨重症肌无力（MG）时神经肌肉接头（NMJ）突触后、前膜的变化及其意义。

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Neurotransmitter release during synaptic transmission is mediated by presynaptic active zone.

在突触前,神经递质释放是由活动区结构所介导的.

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A neurotransmitter must be present within the presynaptic neuron.

突触前神经元必须存在神经递质.

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The defining morphological feature of chemical synapses is the vesicle cluster in the presynaptic nerve terminal.

突触小泡即刻可释放池的结构组织化学突触的定义形态学特征是突触前神经终端小泡群.

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Methods: ELISA was used to test the antibodies to ω-conotoxin binding protein, pre-synaptic membrane ( Prsm) and acetylcholine receptor ( AChR) in sera from 34 MG patients, 20 healthy controls and 21 other neurological disorders ( OND).

方法：用ELISA方法检测34例重症肌无力病人、20例正常人和21例其他神经系统疾病病人血清中抗ω－芋螺毒素结合蛋白、抗突触前膜和抗乙酰胆碱受体抗体。

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A presynaptic nerve fiber; a presynaptic stimulus.

神经对刺激做反应.

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In order to explore the roles of different neurotransmitters in epileptic pathogenesis, the synaptic connections between glutamic acid neurons and GABA neurons in normal rat hippocampus were studied by pre-embedding double labeling immunoelectron microscopy, i. e.

为了探讨神经递质在癫痫发病机理中的作用，用包埋前免疫电镜双标法研究了正常大鼠海马CA1，区谷氨酸（Glu）神经元与GABA神经元之间的突触联系。

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Changes of the ultrastructure: The neurons in the cortex of SHR with the ascending of blood pressure, appeared karyopyknosis, mitochondria swollen, crista lost, the membranes of pre-synaptic and post-synaptic were not clear and the synaptic space was disappeared.

超微结构的改变：SHR大脑皮质内神经元随着血压的升高出现核固缩、线粒体肿胀、嵴消失以及突触前后膜肿胀、突触间隙消失等改变。

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Activity dependent synaptic modification is critical for the development and function of nervous system, the extent and direction of modification may depend on the precise timing of pre-and post-synaptic action potentials.

由动作引起的突触变化对神经系统的生长和功能是十分重要的，其变化的程度和方向是由突触前后动作电位的精确定时引起的。

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Short-term synaptic plasticity includes two forms, short-term enhancement and short-term depression.

短时 程突触可塑性包括 短时 程增强和 短时 程压抑两种类型.

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The soma of CCK-neurons also received afferent synapses from NPY-nerve terminals, in the pre-synaptic element of which NPY and CCK might coexisted.

CCK神经元胞体可接受NPY神经末梢的传入性突触，后者的突触前成分内可能有CCK与NPY共存。

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Objective To explore the expression of synaptic plasticity-associated proteins in hippocampus of rats exposed to EMP.

目的研究电磁脉冲(electromagneticpulse,EMP) 照射后大鼠海马组织中突触可塑性相关蛋白的表达情况.

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A Study on Correlation of Myasthenia Gravis with Seral Acetylcholine Receptor Antibody, Pre-synaptic Membrane Antibody, and Citric Acid Extract Antibody

血清乙酰胆碱受体抗体、突触前膜抗体和胸腺瘤相关抗体与重症肌无力发病关系的研究

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Click Reload or press Ctrl + R to make Synaptic aware of the latest updates.

点击刷新(Reload)或者按下Ctrl+R来使新立得获得最新的软件包信息.

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Probably are two different kinds of the pre-and posts synaptic membranes of adrenergic nerve separately.

可能与肾上腺素能神经突触前膜和突触后膜分别存在的两类不同性质的作用点有关.

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These results suggest that opioid pep -- tides predominantly reduce synaptic transmission in NAcc.

上述结果提示:在伏核内,阿片 肽 的作用在于降低突触传递.

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The N-terminal end of the heavy chain is responsible for binding to specific pre-synaptic neuronal cell receptors and the C-terminal end facilitates internalization.

重链N端能够结合特定胆碱能神经末梢质膜受体，C端主要在内化过程中起作用。

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In this case, the CA-bouton was presynaptic element whereas the VP-bouton served as postsynaptic element.

在这种情况下，含CA的神经终末为突触前成分，含VP的神经终末为突触后成分。

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