1、

A Study on Correlation of Myasthenia Gravis with Seral Acetylcholine Receptor Antibody, Pre-synaptic Membrane Antibody, and Citric Acid Extract Antibody

血清乙酰胆碱受体抗体、突触前膜抗体和胸腺瘤相关抗体与重症肌无力发病关系的研究

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2、

The soma of CCK-neurons also received afferent synapses from NPY-nerve terminals, in the pre-synaptic element of which NPY and CCK might coexisted.

CCK神经元胞体可接受NPY神经末梢的传入性突触,后者的突触前成分内可能有CCK与NPY共存。

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3、

The N-terminal end of the heavy chain is responsible for binding to specific pre-synaptic neuronal cell receptors and the C-terminal end facilitates internalization.

重链N端能够结合特定胆碱能神经末梢质膜受体,C端主要在内化过程中起作用。

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4、

Pre-synaptic test stimulation was given at 0.07 Hz intervals through bipolar stimulating electrodes. EPSCs of layer ⅱ~ ⅳ were recorded by recording electrodes filled with 0.5% biocytin.

通过视皮层的双极刺激电极给予0.07Hz的突触前测试刺激,记录视皮层神经元的PSCs。

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5、

The adenosine exerts its inhibitory effect through activation of both pre-synaptic and post-synaptic adenosine A1 receptors ( A1R's). Thus, A1 receptors play an important role in promoting sleep.

腺苷是通过激活突触前膜和突触后膜腺苷A1受体而起抑制性作用,所以腺苷A1受体在促进睡眠中起重要作用。

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6、

CCK-8S is one of the strongest endogenous anti-opioid substances and suppresses opioid peptides-mediated 'pre-synaptic inhibition' of γ-aminobutyric acid ( GABA) release.

CCK-8S是目前发现最强的内源性抗阿片物质。阿片肽可增强γ-氨基丁酸(GABA)介导的突触前抑制的作用,而CCK-8S可对抗阿片肽的这一作用。

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7、

Changes of the ultrastructure: The neurons in the cortex of SHR with the ascending of blood pressure, appeared karyopyknosis, mitochondria swollen, crista lost, the membranes of pre-synaptic and post-synaptic were not clear and the synaptic space was disappeared.

超微结构的改变:SHR大脑皮质内神经元随着血压的升高出现核固缩、线粒体肿胀、嵴消失以及突触前后膜肿胀、突触间隙消失等改变。

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8、

Methods: ELISA was used to test the antibodies to ω-conotoxin binding protein, pre-synaptic membrane ( Prsm) and acetylcholine receptor ( AChR) in sera from 34 MG patients, 20 healthy controls and 21 other neurological disorders ( OND).

方法:用ELISA方法检测34例重症肌无力病人、20例正常人和21例其他神经系统疾病病人血清中抗ω-芋螺毒素结合蛋白、抗突触前膜和抗乙酰胆碱受体抗体。

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