1、

Hyperplasia of synovium leads to formation of pannus which destroys cartilage bone in the affected joint.

大量增生的滑膜组织会导致血管翳的生成,进而逐渐侵蚀软骨,造成关节的损伤.

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2、

The relative numbers of hematopoietic cells in the synovium were similar in all groups.

所有分组的滑膜中的造血源细胞相对数量是相似的.

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3、

Neuropathic or degenerative joints with hyperemic or inflamed synovium are particularly prone to the development of loose bodies, but they also tend to rapidly reabsorb them.

研究表明伴有充血、炎症滑膜的关节神经痛或者退行性关节炎很可能会产生游离体,但是也容易被吸收。

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4、

Results ( 1) Observation to the tissue form of articular cartilage: In control group, the cartilages were smooth and transparent; no neoplasm, articuli hydrops and synovium hyperplasy.

结果(1)组织形态观察:对照组软骨光滑透明,无赘生物,无明显关节积液及滑膜增生;

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5、

Objective: To know the relation between certain change in knee joint cause synovium exudative inflammation and immunological function changes.

目的:为了解某些膝关节病变引起滑膜炎性渗出与机体免疫功能变化关系,我们对46例膝关节滑膜病变患者检测了血清及滑膜腔积液免疫指标含量变化。

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6、

Conclusions: TGP combined with LEF can delay the progression of RA and provide protection for sclerotin and synovium of joint.

结论:TGP联合LEF治疗RA能稳定病情,对关节滑膜及骨质破坏起到较好的保护作用。

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