1、

The mechanism by which PAF increased pulmonary vascular pressure and resistance was not through inhibition of EDRF synthesis. EDRF did not block PAF-induced changes in pulmonary vascular tone.

PAF增高肺血管压力和阻力的机制不是通过抑制EDRF合成的途径,EDRF不能阻断PAF引起的肺血管张力升高。

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2、

CONCLUSION: Receptor agonists but not nonreceptor agonists-induced EDR are commonly impaired in 4-wk Str-induced diabetic rat aorta, and this defective effect is attributable to the low formation of EDRF/ NO which is related to impaired capacitative Ca2+ entry pathway in endothelium.

结论:在4周链佐星糖尿病大鼠主动脉,受体而不是非受体介导的EDR普遍损伤,其机制与内皮细胞电容性钙内流信号通路受损从而使NO合成减少有关。

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3、

Effect of Endothelium-Derived Relaxing Factor ( EDRF) on Carotid Thrombosis in Rats

内皮舒张因子对大鼠颈动脉血栓形成的影响

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4、

Effect of aggregating platelet on vascular tension and the role of EDRF

聚集的血小板对血管张力的影响及内皮细胞舒血管因子的作用

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5、

These results suggested that IL-8 could dilate blood vessels by stimulating the release of EDRF from endothelium.

结果表明IL-8可以通过促进血管内皮细胞释放一氧化氮而扩张血管。

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6、

Effects of EDRF on the vasoconstrictor action of NE in rat aorta

EDRF对NE引起的大鼠主动脉缩血管效应的作用

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7、

P 6 A was believed to dilate vessels by stimulating the release of EDRF from endothelium.

结果提示肽6A可以通过促进血管内皮释放内皮舒张因子而扩张血管。

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8、

Affection of EDRF/ Nitrous Oxide on pulmonary artery pressure in hypoxic rats

EDRF/NO对缺氧大鼠肺动脉压的影响

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