conclusion the species distribution, molecular electrical property and apparent distribution coefficient of levofloxacin all demonstrated much to be reliant on ph value.

meta-analysis on microbiological outcomes of bacterial infections treated with moxifloxacin versus levofloxacin

莫西沙星与左氧氟沙星治疗细菌性感染的细菌学疗效比较的Meta分析

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objective levofloxacins of various salts were studied for its effect and mechanism on hepatitis b in vitro.

目的研究不同盐的左氧氟沙星体外抗HBV的作用及其机理。

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chinical study of levofloxacin in the treatment of respiratory tract bacterial infections with copd

左氧氟沙星治疗COPD合并肺部感染的疗效观察

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pharmacokinetics and pharmacodynamics of levofloxacin in patients with respiratory tract infections using bayesian forecasting

Bayesian反馈法研究左氧氟沙星在呼吸系统感染患者中药动学及药效学

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hplc determination of levofloxacin in levofloxacin ophthalmic gel bioequivalence of levofloxacin hydrochloride tablets in chinese healthy volunteers

HPLC法测定盐酸左氧氟沙星液状眼用凝胶中左氧氟沙星的含量

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comparison of effect of levofloxacin triple and bismuth quadruple in hp eradication

左氧氟沙星三联与铋剂四联疗法根除HP疗效的比较

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compatibility of levofloxacin injection combined with vitamin c

左氧氟沙星与维生素C注射液的配伍稳定性

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objective to study the effects of cigarette smoke on macrophage metalloelastase ( mmp-12) mrna expression in the rats passively inhaled the smoke and the role of n-acetyl-cysteine ( nac) in the management of copd.

目的：探讨香烟烟雾对被动吸烟大鼠巨噬细胞弹性蛋白酶（MMP-12）表达的影响及N-乙酰半胱氨酸（NAC）在慢性阻塞性肺疾病中的治疗作用。

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the aim of the experiment was to study the lipid peroxidation damage of arsenic and the antagonistic action of n-acetyl-cysteine and vite on the damage induced by arsenic in blood, liver and kidney of rats.

本试验探讨了砷对小鼠血液和肝、肾脂质过氧化损伤及抗氧化剂N-乙酰半胱氨酸（NAC）和VitE对砷致小鼠脂质过氧化损伤的拮抗作用。

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moreover, addition of n-acetylcysteine or glycyrrhizic acid together with acetaminophen reduced the hepatotoxicity of acetaminophen, which illustrated the feasibility of gel entrapment culture of hepatocytes as a hepatotoxicity model to reflect the situation of a hepatoprotective drug in vivo.

此外，N-乙酰半胱氨酸（NAC）与甘草酸（GA）与醋氨酚一起加到培养基中能显著抑制醋氨酚导致的肝毒性，进一步说明凝胶包埋培养肝细胞可以反映保肝药物的临床作用。

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methods: serum creatine kinase was determined by n-acetyl cysteine ( nac) assay in 22 patients with ruptured tubal pregnancies and 35 patients with unruptured tubal pregnancies. fallopian tube tissues were examined by histopathology.

方法：采用N-乙酰半胱氨酸（NAC）法，测定22例破裂型输卵管妊娠患者和35例未破裂型输卵管妊娠患者的血清CK水平，同时对输卵管妊娠组织标本进行病理分析。

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in lps+ nac group, pregnant mice were injected with n-acetylcysteine ( nac) at 30 min before and at 3 h after lps ( 0.2 mg/ kg, ip);

LPS+NAC组在注射LPS（0.2mg/kg，ip）前30min和后3h给予N-乙酰半胱氨酸（NAC）；

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n-acetylcysteine ( nac) is a main part of free thiol in vivo, a precursor of glutathione. and has some effects on scavenging free radicals and protecting cells.

N－乙酰半胱氨酸（NAC）是体内游离巯基的组成部分，既可补充体内谷胱甘肽，又具有清除自由基、保护细胞的作用。

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objective to compare the protective effect of n-acetyl-cysteine ( nac) and dexamethasone ( dex) in acute lung injury ( ali), and to provide a better method in treating ali.

目的比较N-乙酰半胱氨酸（NAC）和地塞米松（Dex）对急性肺损伤（ALI）的防治作用，为临床治疗ALI开辟新的治疗措施，并提供理论基础和实践方向。

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the changes of sod, gpx and mda in 20 patients with pulmonary tuberculosis were observed before and after they had been given n-acetylcysteine ( nac) and anti-tuberculosis treatment for three weeks.

此外，20例肺结核患者在抗结核治疗的同时加用N-乙酰半胱氨酸（NAC）治疗三周，治疗前后均按上述方法测定SOD、GPX、MDA。

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objective: to study the protective effect and mechanism of n-acetyl cysteine ( nac) on the middle cerebral ischemic-reperfusion injury ( ciri) in rats.

目的：研究在大鼠局灶性脑缺血模型中，N-乙酰半胱氨酸（NAC）预防脑组织的缺血/再灌注损伤的效果。

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objective to assess the effect of n-acetylcysteine ( nac, antioxidant) on the airway inflammation in rats with acute and chronic smoking.

目的探讨抗氧化剂N-乙酰半胱氨酸（NAC）对急慢性吸烟大鼠气道炎症的影响。

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32 sd rats were divided into 4 groups: control group, high-fat group, la group ( 0.1%) and nac group ( 0.1%).

32只SD大鼠分成4组，分别是正常饲料对照组、高脂饲料组、高脂饲料+硫辛酸（0.1%）组和高脂饲料+N-乙酰半胱氨酸（0.1%）组。

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methods: male wistar rats were randomly divided into normal control group, cb and emphysema group, em-treatment group and nac-treatment group.

将健康雄性Wistar大鼠随机分成正常对照组、慢支与肺气肿组、红霉素（EM）治疗组、N-乙酰半胱氨酸（NAC）预防及治疗组。

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n-acetylcysteine could inhibit nf-κ b activation, reduce inflammatory reaction and cerebral tissue damage. it might play an important role in the neuroprotective agents in long-term ischemia for 6 and 24 hours.

应用N-乙酰半胱氨酸能抑制核转录因子κB活化，减少炎症反应和脑组织损伤，缩小梗死体积，在6,24h的长时间脑缺血中仍能起到脑保护作用。

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