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The protective effects on cerebral ischemia and ischemia-reperfusion were shown in results.
实验结果表明:QN胶囊对脑缺血及脑缺血再灌注损伤有保护作用。
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冰片注射液对小鼠脑缺血再灌注后学习和记忆行为的影响
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缺血预处理对兔肺缺血再灌注性损伤的蛋白质组学研究
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提示:人参皂甙对犬心肌缺血再灌注损伤后的心肌具有良好的预防作用。
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The IPC 、 PMA protected the myocardium from sustained ischemia-reperfusion injury . Bre.
Bre. 则可消除IPC对心肌的保护作用.
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ICAM-1 may play important roles in myocardial ischemia-reperfusion injury after CPB.
ICAM -1 在CPB后心肌缺血再灌注损伤中起重要作用.
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Study on the mechanism of leukocytes adherence to endothelial cells during ischemia-reperfusion
缺血-再灌注过程中白细胞与内皮细胞粘附机制探讨
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ASI can lower expression of NF-kB and decrease cerebral ischemia-reperfusion injury.
刺五加注射液可下调的NF-kB表达,减轻脑缺血再灌注损伤.
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Objective To study bacterial translocation during mesenteric ischemia reperfusion injury.
目的了解新生儿肠缺血再灌注损伤对细菌移位的影响.
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方法在缺血再灌注前,用低潮气量,低频率的机械通气方法造成动物的呼吸性酸中毒.
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The destruction of BBB is an important mechanism of cerebral ischemia-reperfusion injury.
血脑屏障破坏是脑缺血再灌注(ischemiareperfusion,IR) 脑损伤的重要的病理生理基础.
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线栓法大鼠缺血/再灌注脑损伤模型的改良
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Objective To study the level of nitric NOS in ischemia-reperfusion rat liver.
目的探讨大鼠缺血再灌注肝一氧化氮合酶的变化及其意义.
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CONCLUSION Patients surffer gastrointestinal ischemia reperfusion damage after CPB.
结论CPB术后的胃粘膜出现缺血再灌注损伤.
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Effect of glycogen on calcium of donor liver during ischemia-reperfusion period
缺血再灌注期间供肝糖原水平对供肝细胞内游离钙的影响
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Protective effect of Kaixin powder on kidney in rats with ischemia-reperfusion injury
开心散对肾缺血再灌注大鼠肾脏保护作用研究
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Protective effect of Red Sage Root on lung injury induced by brain ischemia ? reperfusion.
丹参对 脑缺血 再灌注后所致肺损伤的保护作用.
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CONCLUSION DK can protect myocardium against ischemia reperfusion injury.
结论dk对大鼠心肌缺血再灌注损伤有保护作用.
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The role of ATP sensitive potassium channel during ischemia-reperfusion in rabbit heart
ATP敏感性钾通道对家兔缺血/再灌注心肌的作用
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To investigate qualitatively cell death patterns in retinal ischemia-reperfusion.
目的定性研究视网膜缺血-再灌注过程中细胞死亡的方式。
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Research on the creation of global cerebral ischemia-reperfusion injury model in mice
小鼠全脑缺血再灌注损伤模型的研究
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Conclusions Reduced glutathione could protect lung ischemia-reperfusion injury.
结论还原型谷胱甘肽对肺缺血再灌注损伤有保护作用.
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Some studies indicates that iNOS expression increases in renal ischemia-reperfusion injury.
肾缺血再灌注损伤的病理机制非常复杂,研究资料表明,iNOS的过表达参与病理生理过程。
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Conclusion: F 2 can antagonize the arrhythmias induced by ischemia-reperfusion in rat heart.
结论: F2对大鼠心脏缺血再灌注心律失常具有拮抗作用.
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细胞凋亡是肝缺血 — 再 灌注 损伤重要的病理生理特征.
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Objective: Hepatic ischemia reperfusion injury can induces intrahepatic cholestasis.
目的: 肝缺血再灌注损伤可造成肝内胆汁淤积.
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Role of CD4~+ T Cells in the Rat Model of Myocardial Ischemia-Reperfusion Injury
CD4~+T细胞在大鼠心肌缺血再灌注损伤中的作用
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Methods Rats cerebral ischemia-reperfusion model was established with suture emboli method;
方法线拴法制作大鼠脑缺血再灌注模型;
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- 相关词组
- retinal ischemia视网膜缺血
- coronary ischemia冠状动脉缺血;
- white graft ischemia白色皮片缺血
- warm ischemia time热缺血时间
- ocular ischemia syndrome眼缺血综合征
- labyrinth ischemia迷路缺血
- cerebral ischemia脑缺血,大脑局部缺血;
- vertebrobasilar ischemia椎基动脉缺血
- myocardial ischemia心肌缺血
- intermitlent myocardial ischemia间歇性心肌缺血
- ischemia of intestine肠缺血
- uterine ischemia子宫局部缺血
- ischemia of lung肺缺血
- critical limb ischemia严重肢体缺血;
- ischemia of extremity end肢端缺血
- vasomotor labyrinthine ischemia血管运动性迷路缺血
- ischemia reperfusion缺血-再灌注;
- hepatic ischemia肝缺血
- reperfusion injury再灌注损伤
- ischemia of spinal cord脊髓缺血
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