bradykinin

The cellular membrane potential hyperpolarization or depolarization is caused by some merterrials ( such as endothelium-derived hyperpolarizing factor, acetylcholine, bradykinin, intercellular second messager and so on) which take part in the informational communication between VEC and VSMC.

内皮源性超极化因子、乙酰胆碱、缓激肽、第二信使等物质亦可引起内皮细胞或/和平滑肌细胞细胞膜的超极化或去极化，参与血管内皮细胞与平滑肌细胞间的信息传递。

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Carcinoid flush cannot be reproduced by 5-HT infusion though bradykinin injection will produce tolerably close imitation.

5 ― HT的输注不能引起类癌性潮红,相反,舒缓激肽的注入则可得到较近似的阳性结果.

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The kallikrein system, which produces the potent vasodilator bradykinin, is beginning to be studied.

血管舒缓素系统产生强力的血管扩张物质缓激肽, 有人正开始这种研究.

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Can fully inhibit the mice response induced by bradykinin ( 0.5ug/per mouse i.a.) and the ED50 of A-ⅲ is 1.3mg/per mouse.

可完全抑制缓激肽动脉注射所引起的痛反应，其ED（50）为1.3mg/只。

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AIM To investigate the delayed effect of bradykinin on energy metabolism of ischemia reperfusion myocardium in rabbits.

目的通过在体兔心肌缺血再灌注模型探讨缓激肽（BK）对缺血再灌注心肌能量代谢的延迟性影响。

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In Vitro Study of the Mechanism of Bradykinin Selective Opening the Blood Brain Barrier

缓激肽选择性开放血脑屏障的体外探讨

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Carcinoid flush cannot be reproduced by 5-HT infusion though bradykinin injection will produce a tolerably close imitation.

5―HT的输注不能引起类癌性潮红，相反，舒缓激肽的注入则可得到较近似的阳性结果。

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Experimental study of delayed protection of bradykinin on rabbit myocardium

缓激肽对家兔心肌延迟性保护作用的实验研究

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The effect of intrarenal artery injection of bradykinin ( BK, 5 0 μ g/ kg) on multi and single unit recordings of afferent renal nerve activity ( ARNA) was examined in anesthetized 48 rabbits.

在48只麻醉家兔，应用记录肾传入神经多单位和单位放电方法，观察了肾动脉内注射缓激肽（bradykinin，BK，50g/kg）对肾传入神经活动（ARNA）的影响。

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Expression of bradykinin B2 receptor in brain glioma tissue and its clinical significance

缓激肽B2受体在脑胶质瘤患者肿瘤组织中的表达及临床意义

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An Analysis of Influential Factors of Plasma Bradykinin Level in 100 Normal Subjects

100例正常人血浆缓激肽测定及其影响因素的分析

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RESULTS: The ADR induced by benazepril was associated with the aggregation of bradykinin in vivo.

结果: 贝那普利所致不良反应主要表现为咳嗽、皮肤过敏反应等,其与体内缓激肽的聚集有关.

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Bradykinin and angiotensin ( Ang) II are important peptides involved in the regulation of vascular tone.

缓激肽（BK）和血管紧张素II（ANG II）是参与血管紧张性调节的重要的肽。

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NOS activity was significantly increased in bradykinin perfusion group comparing with control group.

缓激肽灌注组的NOS活性显著高于对照组。

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Genetic variation in the promoter region of the β_2 bradykinin receptor gene is associated with essential hypertension in a Chinese Han population

缓激肽β2受体基因启动子区点突变与高血压病的关系

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