audiogenic

Changes of glial fibrillary acidic protein immunoreactivity in medullary visceral zone after acute kindling seizures in audiogenic epilepsy prone rats

听源性惊厥易感大鼠癫痫发作急性期延髓内脏带胶质原纤维酸性蛋白的变化

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Changes of expression of c-fos gene and BDNF in neocortex induced by audiogenic kindling

听源性惊厥点燃诱导新皮质内c-fos和BDNF变化的研究

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Modification of GABA_A receptor subunit mRNA content in audiogenic seizure rat cortex and hippocampus following tolerance to flurazepam

氟西泮耐受听源性惊厥大鼠脑皮质运动区和海马区GABA A受体亚单位mRNA含量的变化

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Electroencephalographic changes during audiogenic seizures in albino rats

听原性癫癎大白鼠在发作时的脑电图变化

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Changes in Expression of Intracerebral Glutamate Transporter GLAST and Regulatory Mechanism of NO in Audiogenic Seizure Rats

听源性癫痫发作大鼠脑内谷氨酸转运蛋白GLAST表达的变化及NO调控机制的研究

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Effects of NE and Pilocarpine Bilateral Inferior Colliculi Injection on Audiogenic Seizures

下丘注射去甲肾上腺素和毛果芸香碱对大鼠听源性癫痫发作的影响

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This paper is to study the dynamic changes in the expression of intracerebral glutamate transporter GLAST in audiogenic seizure rats.

因此，本研究旨在探讨听源性癫痫发作时大鼠脑内谷氨酸转运蛋白GLAST表达的动态变化及L-NNA、L-Arg干预后大鼠癫痫发作的行为学改变及GLAST表达的变化。

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The Effects of Repeated Audiogenic Seizures on Cerebral Glucose Utilization in Audiogenic Seizure Susceptible Rats

反复听源性惊厥对大鼠各脑区葡萄糖摄取率的影响

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The morphological study on NOS-positive neurons in the auditory nuclei and forebrain structures in kindled audiogenic seizure prone rats

听源性惊厥点燃后听觉核团和前脑结构内一氧化氮合酶阳性神经元的形态学观察

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We investigated the seizure score and Ca~ ( 2+)-ATPase activity in brain stem of two kinds audiogenic mice-DBA/ 2 ( D_2) and C_ ( 57)/ B_6 ( B6).

本研究分别观察了DBA/2J（简称D2）和C（57）B4/6（简称B6）小鼠在噪声中癫痫样发作的诱发得分及脑干区Ca~（2+）-ATP酶的活性。

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Expression of synaptophysin p38 in superior colliculus in audiogenic seizures-prone rats

听源性惊厥易感大鼠上丘突触素P38表达的研究

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Audiogenic seizure enhances Cx43 gene expression of astrocyte in rats

听源性惊厥增强大鼠星形胶质细胞Cx43基因表达

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Receptor base on tolerance of the antiepileptic effect to clonazepam in the audiogenic seizure rats

氯硝西泮抗大鼠听源惊厥耐受性的受体学基础

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Researches on relationship between genetically audiogenic seizure mice and the ca~ ( 2+)-atpase activity

遗传性癫痫模型鼠与Ca~（2+）-ATP酶关系的研究

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Change of nitric oxide synthase in the paraventricular nucleus of the audiogenic seizures susceptible rats

一氧化氮合酶在听源性惊厥易感大鼠室旁核的变化

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The results suggest: The elevation of the level of NE in the inferior colliculi reduces the severity of audiogenic seizures, the inhibitory effect of NE on audiogenic seizures is attributable to an action on a receptor similar to the peripheral alpha-2 adrenergic receptor.

实验结果提示：下丘NE水平的提高减弱AS，NE可能是作用于下丘内与外周α2-NE能受体相似的受体而抑制AS的。下丘毒蕈碱样受体的激活对AS起促进作用。

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modification of gabaa receptor subunit mrna content in audiogenic seizure rat cortex and hippocampus following tolerance to flurazepam

氟西泮耐受听源性惊厥大鼠脑皮质运动区和海马区GABAA受体亚单位mRNA含量的变化

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Conclusion A large number of neurons in the PAG of the audiogenic seizures prone rats participated in the process of the audiogenic seizures.

结论听源性惊厥易感大鼠PAG内大量神经元参与了听源性惊厥发作过程。

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The inferior colliculus ( IC) is established as the initiation site within the neuronal network for audiogenic seizures.

一般认为，下丘是听原性惊厥发作神经元网络的启动部位。

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Modification of brain GABA_A receptor subunit α 1,α 3,α 5,γ 2L and γ 2S expression in audiogenic seizure rat following tolerance to clobazam

氯巴占耐受听源性惊厥大鼠脑GABA A受体亚单位表达的变化

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Results After single audiogenic seizure, there were a few dead cells in hippocampus.

结果听源性惊厥单次发作后，海马内未见明显细胞死亡现象；

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These results show that c fos expression induced by audiogenic seizures represents the abnormal neuronal activity which is limited to auditory nuclei, and this abnormal activity may be the basis of audiogenic kindling.

本研究结果表明，听源性惊厥诱导的c－fos表达反映了听觉核团内神经元的异常活动，这种异常的神经元活动可能是听源性惊厥点燃的基础。

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