FDPM

( 2) FDPM lowered the activity of creatine phosphokinase ( CK) and lactic dehydrogenase ( LDH) in serum and myocardium, reduced the occurrence rate of arrhythmia.

二磷酸果糖镁盐降低组织和血清肌酸激酶（CK）与乳酸脱氢酶（LDH）活性，减少心律失常的发生；其作用明显优于1，6一二磷酸果糖、硫酸镁。

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Therefore working on the premise, it is speculated that sodium magnesium fructose diphosphate ( FDPM) which is in part substituted magnesium iron for sodium in fructose-1, 6-diphosphate can take a synergetic effect to protect the tissue and organs of hemorragic shock.

认为镁取代1，6-二磷酸果糖钠盐中部分钠离子衍生为果糖二磷酸钠镁（FDPM）可产生协同作用，从而能更加有效地发挥抗失血性休克及对休克器官组织的保护作用。

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FDPM can decrease the number of NOS-positive cells to inhibit activity of NOS to decrease synthesis of NO and protect rats from ICH.

脑出血后使用FDPM可显著减少NOS阳性神经元，抑制NOS活性，减少NO生成，从而起到脑保护作用。

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Conclusion: FDPM can improve hemodynamics of myocardial ischemia rats induced by pituitrin.

结论：FDPM能改善心肌缺血大鼠的血流动力学。

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These results indicate that FDPM have the protective effect on ischemia reperfusion injury and arrhythmia in myocardium.

表明二磷酸果糖镁盐对缺血心肌有保护作用。

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METHODS: Rat model of intracerebral hemorrhage ( ICH) was induced by collagenase, the timed number of NOS positive cells and the effects of FDPM on it was determined by NADPH histochemical staining.

方法：以胶原酶诱导法制备大鼠脑出血模型，应用NADPH黄递酶组织化学方法，观察脑出血后NOS阳性神经元随时间的变化过程，并观察FDPM对其影响。

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