telomere

Heat shock protein 90 which affects on telomerase and telomere length, which may be related to one of molecular biological mechanism of spleen Yin deficiency syndrome.

参与细胞信号转导的热休克蛋白90，其作用于端粒酶而影响端粒的长度，基于蛋白质组学理论的端粒长度缩短可能是脾阴虚证发生的分子生物学机制之一。

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Overall, the study participants had an average telomere loss of 21 nucleotides ( structural units ) per year.

总的来说, 该项研究的参与者每年平均损失21个核甘酸 ( 结构单位 ).

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Telomere, telomerase and hepatocyte immortalization

端粒和端粒酶与肝细胞的永生化

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Telomerase is a ribonucleoprotein complex whose function is to add telomere repeats to chromosomal ends to make its stable and cell immortalization.

端粒酶是核糖核蛋白复合体，其丰要功能是增加染色体末端端粒的不断重复以维护它的稳定性和细胞的恶性增殖。

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Telosome: A Complex Regulating Telomere

端粒体&调节端粒的多聚复合体

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Age-related changes in telomere of brain cell in SAMP 10

快速老化SAMP10小鼠脑细胞端粒增龄性变化

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Result: Compare with the normal control, the activity of GSH-PX in the liver of rat model with spleen Yang deficiency syndrome was lower and telomere length was shorten.

结果：与正常对照组相比，脾阳虚证的肝组织中的谷胱甘肽过氧化物酶（GSH-PX）活性明显降低（P

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Considering the risks of telomere-extension therapy, he thinks that scientists have bigger fish to fry.

因为延长端粒疗法具有危险, 他认为还有更大的问题需要科学家去解决.

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Telomere is at the end of the chromosome in eukaryotic cells and protects chromosomes.

摘要端粒位于真核细胞染色体末端,起保护染色体末端的作用.

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Effect of Cynomorium songaricum polysaccharide on telomere length in blood and brain of D-galactose-induced senescence mice

锁阳多糖对D-半乳糖衰老小鼠血细胞和脑细胞染色体端粒长度的影响

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Every time the Chromosome divides, every time one cell divides into two. Pieces of the ends of the Chromosome, the telomere, get broken off.

每次染色体分裂，一个分裂成两个，染色体的尾部，也就是端区，就会发生断裂。

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The telomere is like a cellular clock, because every time a cell divides, the telomere shortens.

端粒就像是细胞的时钟, 因为在每次细胞分裂时它都首先要缩短.

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Conclusion: Shortening of telomere may be one of pathomechanism of spleen Qi deficiency syndrome.

结论：提示端粒长度缩短可能是脾气虚证的病理机制之一；

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Therefore, TRF 2 may protect chromosome ends by maintaining the correct structure at telomere termini.

因此, TRF2可保护染色体末端保持正确的结构,端粒末端.

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Telomere locates on the terminal of DNA containing repeat DNA sequence.

端粒是位于染色体末端的串联重复核苷酸序列.

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Telomere/ Telomerase System and Human Hematopoietic Cells

端粒/端粒酶系统与人类造血细胞

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Experimental Study on the Change of Telomere Length in the Liver of Rat Model with Spleen Yang Deficiency syndrome

脾阳虚证大鼠肝组织中端粒长度变化的实验研究

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Conclusion Telomere reduction and telomerase activation are critical factor during oncogenesis.

结论端粒缩短并激活端粒酶是涎腺肿瘤形成的重要因素.

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Conclusion: The shortening of telomere length may be related to the free radicals damage with spleen Yang deficiency syndrome.

结论：在脾阳虚状态下，肝细胞端粒长度缩短可能与自由基损伤有关。

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Can telomere maintenance be eliminated effectively ( and selectively ) enough by more tractable approaches?

端粒维持可以由足够有效地 ( 或选择性地 ) 更易控制的途径而有效地被排除 吗 ?

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We summarize the present research of telomerase and telomere and their applications on ocular disease.

在此,我们就端粒、端粒酶研究现状及其与眼科疾病的关系和应用进展作一综述.

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The changes in the telomeres found in lizards that have experienced a close call adds to the evidence that environmental stress has negative effects by eroding telomere length.

在遭遇过险情的蜥蜴体内的端粒所发生的变化进一步证明了生存环境的压力会给蜥蜴带来负面的影响，使其端粒长度变短。

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Telomerase system is the genetic name of telomerase and telomere.

端粒酶系统是端粒和端粒酶的总称.

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The findings held even after the researchers took into account other factors that could affect telomere length, including the women's chronological age, body mass index and smoking habits.

即便研究人员们考虑了其它能影响端粒长度的因素（比如她们的实际年龄、身体质量指数和吸烟习惯），这些发现依旧站得住脚。

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Telomere does not have any password function, which is like a tall paper hat at the head of chromosomes.

端粒本身没有任何密码功能，它就像一顶高帽子置于染色体头上。

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Researchers estimate that telomere maintenance activity occurs in about 90 % of human cancers.

研究人员估计,90%的癌症中端粒一直处于活跃状态.

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ALT and TA mechanisms are dominant maintenance mechanisms of the telomere length.

端粒长度的维持机制主要有ALT机制和TA机制.

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Telomere dynamics of leukocytes in bone marrow with leukemia and myelodysplastic syndromes by flow cytometry

流式原位杂交法检测白血病和骨髓异常增生综合征患者端粒长度的研究

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Objective To evaluate the role of telomere and DCC in tumor transformation and progression.

目的　分析端粒长度及DCC基因 mRNA 表达在大肠癌及腺瘤发生发展中的作用.

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Several factors, such as defects in chromosome segregation and DNA damage response, failure of cell cycle checkpoint, telomere dysfunction, may cause chromosomal instability.

多种细胞机制功能障碍将导致染色体不稳定性，包括染色体分离、DNA损伤反应、细胞周期检查点、端粒功能等。

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