atheroma的例句大全

Expression of chlamydia pneumoniae specific antigen in human coronary artery atheroma tissues

冠状动脉瘤组织中肺炎衣原体特异性抗原的表达

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The expression of MMP-8 in different cell types of atheroma was detected by immunohistochemical staining.

用免疫组化的方法测定MMP8在粥样斑块不同细胞成分中的表达。

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At the22th week, atheroma and calcification were present.

实验22周时可见粥样斑块形成及管壁钙化。

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There was a significant difference in positive rate of Cpn-Ag between coronary artery atheroma specimens and normal artery specimens ( P0.01).

粥样硬化冠状动脉瘤标本和正常冠状动脉标本外膜下和/或斑块内Cpn-Ag检测结果间差异有显著性意义（P0.01）。

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Compensatory enlargement of human coronary arteries during progression of atherosclerosis is unrelated to atheroma burden: Serial intravascular ultrasound observations from the REVERSAL trial

动脉粥样硬化进展时冠状动脉代偿性扩张与粥样斑块负荷无关：REVERSAL试验中连续血管内超声观察结果

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The duration from therapy of thrombolysis to clinical syndrome ameliorated was more prolonged in groups of atheroma thrombosis and severe stenosis ( P < 0.05).

颈动脉粥样硬化性完全梗塞和严重狭窄组病例溶栓起效时间明显推迟（P＜0.05）。

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Background-Acute coronary syndromes ( ACS) are complications of atherosclerotic vascular disease that are triggered by the sudden rupture of an atheroma.

研究背景：急性冠脉综合征是冠状动脉粥样硬化斑块突然破裂所触发的一组临床症候群。

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Methods The Cpn-Ag was detected by direct immunofluorescence using self-made monoclonal antibody of Cpn for 50 archival paraffin-embedded coronary artery atheroma specimens and 20 normal coronary artery specimens.

方法采用自制肺炎衣原体单克隆抗体的直接微量免疫荧光法检测50例冠状动脉瘤标本和20例正常冠状动脉标本中Cpn-Ag的表达。

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Objective To probe the roles of the ligands of peroxisome proliferator activated receptor α ( PPAR α) and PPAR γ in the pathogenesis of atheroma lesion.

目的探讨PPARα和γ配体在动脉粥样硬化病变局部的作用。

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Conclusion After 12w endurance training, the higher levels of TC in plasma and MDA in vessele wall in ApoE-/-mice did not promote atheroma plaque development.

结论：12周耐力运动后ApoE-/-鼠血浆TC和血管壁MDA水平升高，但TC的升高没有促进动脉粥样硬化斑块的发展。

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Studies on Role of Serum β-Lipoprotein in the Pathogenesis of Atheroma ⅱ. Incorporation of~ ( 131) I-Labelled Rabbit Serum β-Lipoproteins into Rabbit Aorta

血清β脂蛋白在动脉粥样硬化斑块形成中的作用&Ⅱ.~（131）Ⅰ标记兔血清β脂蛋白参入主动脉壁的研究

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Many basic and clinical studies in recent years have reported atheroma/ atherosclerosis as a disease driven by a chronic inflammation in blood vessels, and inflammation plays a critical role during the formation, evolution and breaking of atherosclerotic lesion.

近年大量的基础和临床研究表明，动脉粥样硬化是一种慢性炎症改变，炎症在动脉粥样硬化斑块的形成、演变及破裂过程中起着至关重要的作用。

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The maximum plaque thickness, the average plaque thickness, the degree of aortic stenosis and the proportion of the intimal circumference occupied by atheroma of 3 groups were different from each other significantly ( P < 0.01).

光镜下动脉粥样硬化斑块多参数分析显示，斑块最大厚度、斑块平均厚度、管腔狭窄度和斑块所占周径比值4个指标各组间两两比较差异有显著性（P

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Methods The human PBMC was separated and Cpn-Ag was measured by direct immunofluorescence ( DI) test, at the same time, the expression of Cpn-Ag was detected by DI using self-made Cpn-McAb for coronary artery atheroma specimens and normal coronary artery specimens.

方法采用自制Cpn-McAb的直接免疫荧光（DI）法检测颈动脉粥样硬化和健康体检者PBMC中Cpn-Ag及冠状动脉瘤和正常冠状动脉标本中Cpn-Ag的表达。

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The atherosclerotic plaques in UA and AMI patients were mainly the atheroma ( unstable plaque) with large necrotic core (> 40%), thin fibrous cap, less smooth muscle cells and abundant foamy cells.

UA和AMI病例中斑块主要为不稳定斑块，病理特征为脂质坏死中心大（大于40%），纤维帽薄，平滑肌细胞少而泡沫细胞多。

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Cholesterol clefts are numerous in this atheroma.

有大量的胆固醇结晶.

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This is a high magnification of the aortic atheroma with foam cells and cholesterol clefts.

高倍显示动脉粥样硬化时的胆固醇结晶和泡沫细胞.

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A few dark blue inflammatory cells are scattered within atheroma.

一些暗蓝色的炎细胞散在分布于粥样硬化病灶内.

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This microscopic cross section of the aorta shows a large overlying atheroma on the left.

动脉纵切镜下显示左侧有大的动脉粥样硬化病灶.

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This high magnification of the atheroma shows numerous foam cells and an occasional cholesterol cleft.

动脉粥样硬化高倍镜显示多量泡沫细胞,偶见胆固醇结晶(棱状空隙).

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