ICAM的例句大全

Continuous ICAM 1 expression might be related to the stenosis of eustachian tube.

咽鼓管狭窄可能是粘膜ICAM- 1持续表达的原因之一.

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Immunohistochemical and biochemical techniques were used to detect myocardial ICAM-1 and myeloperoxidase ( MPO ).

采用免疫组织化学和生化法检测心肌ICAM -1 的表达及髓过氧化物酶 ( MPO ) 水平.

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Results: 1 . The protein and mRNA of ICAM-1 was expressed at low levels on nomoral TEC.

结果: 1. 正常大鼠肾小管上皮细胞ICAM -1 蛋白质和mRNA低水平表达.

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The expression of ICAM-1 mRNA can be inhibited when treated with GCV and PFA ( P < 0.05 ).

GCV和PFA可 抑制HCMV感染诱导的ICAM -1 表达的上调 ( P<0.05 ).

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Asarinin could also decrease the level of ICAM-1 and VCAM-1 in the allografts.

CsA组 、细辛脂素组和混合药物组移植心局部ICAM -1 和VCAM -1 含量较阳性对照组明显减少.

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Results showed that ICAM-1 immunoreaction was negative or weak in normal control group.

结果显示正常对照组大鼠脑血管ICAM -1 免疫组织化学显色为阴性或弱阳性反应.

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ICAM-1 may play important roles in myocardial ischemia-reperfusion injury after CPB.

ICAM -1 在CPB后心肌缺血再灌注损伤中起重要作用.

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Objective To investigate the change and clinical value of C-peptide ( CP ), intercellular adhesion molecular-1 ( ICAM-1 ), insulin resistance of traumatic patients.

目的探讨创伤患者急性期C肽 ( CP ) 、 细胞间黏附分子 -1 ( ICAM -1 ) 、 胰岛素抵抗(IR)的变化特点及其临床意义.

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Each group was subdivided into 0-hour IR, 3-hour IR and 6-hour IR subgroups. The rats were scarified at corresponding time point under anesthesia. The expression of ICAM-1 was tested by using immunohistochemistry, and the activity of MPO by chromatometry.

各组均又分为再灌注0,3,6h组，再相应时间点麻醉状态下处死大鼠取脑，用免疫组织化学方法检测缺血脑组织细胞间黏附分子1表达，用比色法测定缺血脑组织髓过氧化物酶活性。

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Conclusion JNK/ SAPK may have been aberrantly activated in MSPGN, which may involve in the overexpression of ICAM 1 in MSPGN.

结论MSPGN的肾小球系膜细胞的JNK/SAPK存在异常活化，这种异常活化可能介导了ICAM-1的过度表达。

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Aprotinin could downgrade the expression of ICAM-1 of TNF α activating endothelial cells and prevent the loss of β-catenin of the cells, protect the adherens junctions of endothelials cells.

抑肽酶可抑制TNFα活化的人脐静脉内皮细胞ICAM-1的表达，可避免β-catenin的丢失。

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After TCR/ CD3 complex cross-linking MHC/ peptide, LFA-1, expressed on T cell increases a higher affinity and avidity for ICAM-1 rapidly. LFA-1 is a key molecule in formation of the immune synapse.

T细胞表面LFA1对ICAM1的亲和力及亲合力在TCR/CD3交联MHC/抗原肽后迅速上调，并参与T细胞免疫突触的形成。

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Conclusion High or low expression of adhesion molecules ICAM-1 、 CD_ 44 V6 、 CD_ 28 、 E-cad and CEA plays a certain role in organic selection of gastric carcinomatous metastasis.

结论粘附分子ICAM-1、CD44V6、CD28、E-cad、CEA表达的增强或减弱在胃癌转移器官选择性中起到一定的作用。

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The expression of ICAM-1 mRNA can be inhibited when treated with GCV and PFA ( P < 0.05 ).

GCV和PFA可 抑制HCMV感染诱导的ICAM -1 表达的上调 ( P<0.05 ).

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Conclusion Melatonin can protect small intestinal mucosa barrier through decreasing the shift of intestinal bacteria endotoxin, lowering cell factor level in plasma and inhibiting ICAM-1 expression in intestinal tissue after obstructive jaundice.

结论褪黑素通过减少肠道细菌内毒素的移位，降低血浆中细胞因子水平，抑制梗阻性黄疸后肠组织ICAM-1的表达，保护小肠黏膜屏障。

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The staining of ICAM-1 in the wall of pustule, dermis and vascular in nipple and keratinized cell was positive, whereas in the normal skin of the controls was negative.

PP患者皮损组织的脓疱壁、真皮浅层和乳头内血管及表皮角质形成细胞ICAM-1染色呈阳性；

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The Pathogenetic Significance of CD_4~+, CD_8~+ Cell and ICAM-1 Expression Changes in Peripheral Blood of NZB/ NZW F1 Mice Lupus-like Change

NZB/NZW F1小鼠外周血CD4~+CD8~+细胞及ICAM-1表达变化对狼疮样改变的发病学意义

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Conclusion: QKT has good antivirus effect, and it may be the possible mucosal immunological mechanism that QKT can increase IgA and reduce ICAM-1's express in the intestine.

结论：前胡咳痰汤有明显的抗病毒作用，能增加肠道IgA并抑制肠道ICAM1表达可能是其药理作用的黏膜免疫机制。

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Approaching the effect of LNR overexpression on the expression of ICAM-1 in cholangiocarcinoma and the molecular mechanism.

LNR过表达对胆管癌细胞ICAM-1表达的影响及其机制。

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The expression of E-selectin and ICAM-1 on BAEC was detected by cell-ELISA.

应用细胞ELISA方法，检测BAEC表面的E选择素和ICAM-1的表达；

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