lipopolysaccharide

Lipopolysaccharide Inhibits Release of S-100b Protein from Cultured Rat Anterior Pituitary Cells

脂多糖抑制离体培养大鼠垂体前叶细胞释放S-100b蛋白

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With gram-negative organisms, depletion of the lipopolysaccharide layer with EDTA greatly facilitates TO uptake.

对革兰氏阴性的生物体, 用EDTA损耗多脂糖层很容易促进TO上调.

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Lipopolysaccharide ( LPS), a component of the outer membranes of Gram-negative bacteria, is a potent activator of macrophages and a major inducer of endotoxin shock.

脂多糖（1ipopolysaccharide，LPS）是革兰氏阴性菌外膜的主要成分，能够强烈激活巨噬细胞，是引起内毒素性休克的主要原因。

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Lipopolysaccharide induced Fos expression of catecholaminergic neurons in medullary visceral zone

延髓内脏带儿茶酚胺能神经元对LPS免疫刺激的Fos表达

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In recent years, the relationship among atherosclerosis, Gram-negative bacteria and lipopolysaccharide has become a research hotspot.

近年来，革兰氏阴性菌感染与革兰氏阴性菌细胞壁重要成分脂多糖（LPS）与AS的关系成为研究的热点。

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Conclusion: Extra estrogen could prevention of acute lung injury induced by lipopolysaccharide.

结论: 外源性雌激素对脂多糖诱导的急性肺损伤有保护作用.

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Study of extracting lipopolysaccharide from rice bran with membrane technology

膜分离技术提取米糠脂多糖的研究

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Among these factors, Gram-negative bacteria infection occupy important status in clinic. The Lipopolysaccharide ( LPS), main composition of endotoxin of its cell wall can cause many kinds of organism damage.

在构成第二次打击的各种致伤因素中，革兰氏阴性菌感染在临床中占有重要地位，其细胞壁内毒素的主要成分脂多糖（Lipopolysaccharide，LPS）可引起多种脏器损伤。

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Autoradiograph assay of p38 MAPK ( mitogen activated protein kinase) activity was established, and applied in measurement of p38 MAPK activity of vascular endothelial cell stimulated by lipopolysaccharide ( LPS).

建立p38丝裂原活化蛋白激酶（Mitogen activated protein kinase，MAPK）放射自显影激酶活性测定方法，并应用于血管内皮细胞中p38MAPK活性动态变化的研究。

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Objective: To observe the direct injury of lipopolysaccharide ( LPS) on cultured human umbilical vascular endothelial cells ( HUVEC) and the effect of IL 10 on it.

目的：观察脂多糖（LPS）对体外培养的人脐静脉内皮细胞（HUVEC）凋亡和坏死的直接作用以及IL10对其影响。

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Lipopolysaccharide ( LPS) is the major component of the outer membranes of the walls of gram negative bacteria that protects bacterial cells against antibacterial agents.

脂多糖（LPS）是革兰氏阴性细菌外膜的主要组成部分。它能通过对抗抗菌剂来保护细菌的细胞壁。

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Experimental Study of Cerebral White Matter Damage in Premature Rats Induced by Lipopolysaccharide

脂多糖致早产鼠脑白质损伤的实验研究

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The avidity of IgG antibody against P. gingivalis lipopolysaccharide ( LPS) was measured by diethylamine dissociation ELISA.

牙周治疗前及治疗3个月、6个月和12个月后常规临床检查、记录牙周附着水平，通过硫氰酸铵分离试验测定血清抗牙龈卟啉菌脂多糖（LPS）的抗体亲和力。

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Acute Injuries in Multiple Tissues in a Rat Heatstroke Model Induced by Co-stress of Lipopolysaccharide and Heat

过热复合脂多糖致热射病大鼠动物模型多器官急性损伤的实验研究

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The Association between Lipopolysaccharide Receptors Polymorphism with Inflammatory Response and Host Susceptibility to Postburn Sepsis

内毒素受体基因多态性与炎症反应及烧伤脓毒症易感性关系的研究

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Effect of TNF α monoclonal antibody on tissue lipopolysaccharide binding protein mRNA expression in rats after thermal injury

肿瘤坏死因子-α单抗对烫伤大鼠组织脂多糖结合蛋白mRNA表达的影响

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Effect of atorvastatin on lipopolysaccharide-induced expression of C-reactive protein in human pulmonary epithelial cells

阿托伐他汀对脂多糖诱导下人肺上皮细胞C反应蛋白表达的影响

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Modification of membrane lipid of human umbilical vein endothelial cells after direct lipopolysaccharide injury

脂多糖直接损伤血管内皮细胞后内皮细胞膜脂的修饰

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Effect of rapamycin on Fos and nNOS expression in the immunity correlative neurons challenged with lipopolysaccharide in rat

免疫抑制剂雷帕霉素对LPS免疫激发大鼠脑内免疫相关核团神经元Fos和nNOS表达的影响

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Lipopolysaccharide ( LPS) is a toxic component of cell walls of Gram-negative bacteria and is widely present in the gastrointestinal tracts of humans and animals.

脂多糖（lipopolysaccharide，LPS）是革兰氏阴性菌细胞壁组成成分，也是革兰氏阴性菌被人类和其他动物消化后释放的毒性成分，广泛存在于消化道中。

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The Influence of the Acidic Components of Aubergine Root to the Murine Peritoneal Macrophages NF-κ B Activated by Lipopolysaccharide

茄根酸性组分对脂多糖诱导的小鼠腹腔巨噬细胞核因子κB表达的影响

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Effects of cigarette smoke and lipopolysaccharide on expression of heat shock protein 70 in lung tissues of rats

香烟烟雾和脂多糖对大鼠肺组织热休克蛋白70表达的影响

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According to capsular polysaccharide and lipopolysaccharide antigen, APP could be divided into 15 serovars, the prevalence and virulence of each serovar were different. Serovar 1,2,3 and 7 are prevalent in China.

根据APP荚膜多糖（CPS）和脂多糖（LPS）抗原性的不同可将APP分为15个血清型，每个血清型的流行地区和毒力有所差别，我国主要流行1、2、3和7型。

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Lipopolysaccharide ( LPS, endotoxin)-the essential pathogenetic component of gram-negative bacterium-was injected into artery. LPS reached brain tissue and caused pathological change.

本实验将革兰氏阴性菌的主要致病成分一一脂多糖（Lipopolysaccharide，LPS）即内毒素（endotoxin）注入实验动物动脉血流，使其到达脑组织，引起病理改变。

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Analysis of the early and late gene expression of lipopolysaccharide activated macrophages by cDNA microarray

内毒素活化巨噬细胞早期和晚期基因表达的cDNA芯片分析

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The main component of the endotoxin, lipopolysaccharide ( LPS) derived from the outer membrane of Gram-negative bacteria cell wall is composed by the core oligosaccharide, specific polysaccharide and lipid A which is the active site and the center virulence of LPS.

脂多糖（LPS）是内毒素的主要成分，来源于革兰氏阴性菌细胞壁的外膜，由核心寡聚糖、特异性多糖与类脂A组成，其中类脂A是LPS的活性部位以及毒力中心。

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Selective impairment of lipopolysaccharide on antioxidant system in rat hepatocarcinogenesis induced by thioacetamide

脂多糖对化学诱癌大鼠抗氧化系统的选择性损害作用

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Protective Mechanism of Chloroquine on Lipopolysaccharide-Induced Acute Lung Injury in Rat

氯喹对脂多糖致急性肺损伤大鼠的保护性作用

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Effect of IL-10 on the Direct Injury Induced by Lipopolysaccharide in Cultured Human Umbilical Vascular Endothelial Cells

白细胞介素-10对脂多糖直接诱导的血管内皮细胞损伤的影响

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CD14 is a receptor of lipopolysaccharide ( LPS) of gram negative bacteria and expresses on membrane surface of phagocytes.

CD14作为革兰氏阴性菌细胞壁脂多糖的受体，表达在吞噬细胞膜表面上。

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