Method Root canals of chronic pulpitis and ( or) apicitis were filled using the FR paste.

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Conclusion: The method to establish a rat pulpitis induced by LPS was feasible.

结论：LPS法诱导大鼠实验性牙髓炎简便可行。

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Unmineralized reparative dentin, odontoblast-like cells, human dental pulp cells and endothelial cells of the blood vessels.

在牙髓炎人牙中，LIM矿化蛋白1主要分布于前期牙本质、成牙本质细胞、未矿化的修复性牙本质、成牙本质细胞样细胞、牙髓成纤维细胞及血管内皮细胞。

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METHODS: 212 molars were diagnosed as acute pulpitis or chronic pulpitis, and randomized block designed into two groups: The experiment group: 104 cases were devitalized by devitalizing paste without Arsenic.

方法：将212例临床诊断为急性牙髓炎或慢性牙髓炎的磨牙随机分为2组，实验组104例采用无砷失活剂（PD，瑞士产）失活；

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Observation Effect Case of the Acute Pulpitis Cured with One-shot Root Canal

一次性根充法治疗急性牙髓炎130例的疗效观察

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Moreover, the proportion of the five microorganisms in the total bacteria is small, so there may be still other pathogenic microorganisms involved in the occurrence and development process of retrograde pulpitis.

Aa、Pg、Pi、Tf和Td五种微生物在总菌中所占的构成比不大，提示可能尚存在其他的优势致病微生物参与逆行性牙髓炎的发生发展过程。

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Conclusion: The results suggest that GDNF may take part in regulating nerve-sprouting reactions during dental restorative process.

结论：牙髓炎修复期GDNF在尾侧亚核F的表达增高，提示GDNF可能参与炎症牙髓神经修复再生过程，对神经末稍再生发芽有促生长作用。

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The Expressions of Intercellular Adhesion Molecule-1 in Experimental Pulpitis of the Rat

细胞间粘附分子-1在大鼠实验性牙髓炎中的表达及意义

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Study on Oxygen-Derived Free Radicals of the Inflammatory Pulp Tissue

牙髓炎组织中氧衍生自由基的研究

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Aim: To investigate the change of SP and CGRP in the pulpal tissue with acute pulpitis and after opening of pulp chamber.

目的：比较和分析急性牙髓炎和牙齿开髓刺激后，牙髓组织中P物质（SP）、降钙素基因相关肽（CGRP）的变化。

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Objective To investigate the transmission signal of the experimental pulpitis pain.

【目的】了解实验性牙髓炎疼痛信号在中枢的传导特点。

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Experimental Study on Pathogenetic Mechanism of Lipopolysaccharide ( LPS) on Pulpitis

内毒素脂多糖在牙髓炎症中致病机理的实验研究

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A study on NOS in trigeminal ganglion and trigeminal spinal caudal subnucleus during pulpitis

牙髓炎症中三叉神经节和三叉神经脊束核尾侧亚核NOS的变化

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Methods: Rat pulpitis model and immunohistochemical technology.

方法：大鼠牙髓炎模型和免疫组织化学技术。

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So it is important to study anti-LPS methods to lessen the toxic effects of LPS on pulp cells for the treatment of pulpitis and protection of vital-pulp.

因此研究拮抗LPS的治疗措施，降低LPS对牙髓细胞的毒性作用，对牙髓炎的防治和活髓的保存具有重要意义。

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[ AIM]: To establish a model of rat endotoxic pulpitis induced by Lipopolysaccharide ( LPS);

[目的]：运用内毒素（LPS）诱导法建立大鼠牙髓炎实验模型；

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The Clinical Evaluation of Treatment of 127 Cases Molar Acute Endodontitis

127例磨牙急性牙髓炎治疗的临床评价

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Influence of mental intervention on dental anxiety in adult with pulpitis

心理干预对成人牙髓炎患者牙科焦虑症的影响

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RESULTS: No IL-8 was detected in the samples from normal pulp, but significant amount of IL-8 appeared in inflamed pulp tissues, and the level of IL-8 in exudates of acute stage of pulpitis was higher than that of chronic stage ( P < 0.01).

结果：正常牙髓组织渗出液中检测不到IL-8，而炎症牙髓组织渗出液中均有较高水平的IL-8（0.33～1.0μg/L），且在急性牙髓炎中的水平高于慢性牙髓炎（P<0.01）。

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Clinical curative effect of single-appointment root canal treatment in patients with molar pulpitis

一次法根管充填治疗后磨牙牙髓炎的临床疗效观察

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Establishment of an Experimental Model of Rat Molars Pulpitis Induced by Lipopolysaccharide

大鼠磨牙实验性牙髓炎动物模型的建立

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