The change of endogenous opiate peptides and monoamines in cerebral tissue after cerebral ischemia

1、

Expression of inflammatory mediators and ICAM-1 in lung tissue after gut ischemia reperfusion in rats

肠缺血-再灌注大鼠肺组织炎症介质及ICAM-1表达的变化及意义

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2、

Therefore thrombolytic therapy combined with preventing inflammatory response might contribute to relieving cerebral tissue damage after ischemia.

因此溶栓同时联合抑制炎症反应，可能对减轻缺血脑组织的损伤有益。

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3、

Tissue injury from ischemia and reperfusion causes significant morbidity and mortality in cerebrovascular diseases.

目的：缺血/缺氧性脑损伤是脑血管病的主要致病机制之一，也是其高死亡率和高致残率的重要原因。

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4、

Conclusion GST emergency treatment has obviously protective effect on the nerve tissue of cerebral ischemia injury.

结论GST应急治疗对缺血性脑损伤的神经组织具有明显的保护作用。

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5、

The expression of telomerase in myocardial tissue after myocardial ischemia in rat

大鼠心肌缺血后心肌组织端粒酶的表达变化

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6、

It can relieve the injury on brain tissue after ischemia, shorten the cerebral infarction volume, and prolong the time window of thrombolysis therapy in rats with cerebral infarction during the link of inflammation injury.

可减轻缺血后脑组织的损伤，缩小脑梗死体积，在炎症损伤环节上可延长大鼠脑梗死的溶栓治疗时间窗。

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7、

Objective To study the cyclooxygenase-1 ( COX-1) and cyclooxygenase-2 ( COX-2) messenger RNA ( mRNA) expression in rat brain tissue of ischemia/ reperfusion.

目的研究比较脑缺血及再灌注期环加氧酶异形体（COX-1和COX-2）基因的表达及其意义。

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8、

Acupuncture can act as a pretreatment method of heart muscle ischemia, inducing the tolerance of heart muscle ischemia, and relieve the impairment of cardiac muscle tissue after ischemia.

针刺可以作为一种心肌缺血的预处理手段，诱导心肌缺血耐受，减轻缺血后心肌组织的损伤。

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3 ′-Daidzein Sulfonate Sodium ′ s Effect on NO, ANG ⅱ and ANP Levels in Isolated Myocardial Tissue with Ischemia/ Reperfusion Injury

3′-大豆苷元磺酸钠对离体心肌缺血再灌注时NO ANGⅡ及ANP水平的影响

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10、

Conclusion SATP can protect the cerebral tissue from ischemia injury by decreasing the η B and η P and platelet aggregation and adhesion functions.

结论：SATP可能通过降低血液粘度及抑制血小板粘附和聚集功能对大鼠实验性脑缺血产生保护作用。

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11、

Objective: To investigate the effects of carbachol on local inflammation in gut tissue during ischemia/ reperfusion ( I/ R).

目的：研究拟胆碱药卡巴胆碱对缺血再灌注肠道局部炎症反应的影响及意义。

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12、

Acupuncture preconditioning can improve tolerance cardiac muscular tissue to ischemia, increase survival ratio of cardiomyocyte and improve prognosis. 2.

结论：①针刺预处理可以提高心肌组织对缺血的耐受能力，增加心肌细胞的存活率，改善预后。

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13、

Conclusion: The growth of vascular endothelial cells could be inhibited by high concentration of glucose, while Angelica could protect the vascular endothelial cells exposure to high dose of glucose, which might be one of the mechanisms of Angelica treating diabetes associated with tissue ischemia diseases.

结论：高浓度葡萄糖可损伤血管内皮细胞，当归可保护高浓度葡萄糖下血管内皮细胞，这可能是当归治疗糖尿病并发组织缺血性疾病的机制之一。

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14、

Conclusions HIF-1 α antisense oligonucleotide can inhibit angiogenesis and circulating endothelial progenitor cell mobilization in rat hind limb ischemia through regulating role of the VEGF. HIF-1 α is a mobilization factor of EPCs during tissue ischemia.

结论全硫代修饰HIF-1α反义寡核苷酸可抑制大鼠缺血肢体血管生成和因缺血引起的EPCs的动员，其机制可能是通过VEGF起作用，HIF-1α是组织缺血时EPCs一个动员因子。

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15、

Studies have shown that the decreasing of blood cell deformability cause microcirculation disorders and local tissue ischemia and hypoxia in diabetes and a variety of diseases, it is the main reason for diabetes complications can happen easily.

研究表明，糖尿病、高血压等[4-5]多种疾病红细胞变形能力下降，引起微循环灌注障碍和局部组织缺血缺氧，是糖尿病并发症极易发生的主要原因。

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Objective To study the effect of hypoxia-inducible factor-1 α( HIF-1 α) on angiogenesis and endothelial progenitor cell ( EPCs) mobilization during tissue ischemia.

目的探讨低氧诱导因子1α（HIF-1α）在组织缺血时血管生成及内皮祖细胞（EPCs）动员中的作用。

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17、

Objective To examine whether oral folate supplementation would rescue hypercholesterolemia ( HC)-related impairment of angiogenesis in response to tissue ischemia.

目的检验口服叶酸能否改善高脂血症（HC）对缺血组织血管新生的抑制。

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OBJECTIVE: Vascular bypass graft is an important method to reconstruct vascular function when tissue ischemia caused by arterial stenosis or occlusion.

目的：动脉狭窄或闭塞导致的组织缺血坏死应用血管旁路移植是重要的功能修复重建方法。

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19、

Aminoguanidine Improved the Angiogenesis Induced by Tissue Ischemia in Diabetic Mice

氨基胍改善糖尿病小鼠组织缺血后血管新生障碍

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20、

The increased endothelial permeability is the prevalent and important characteristics of inflammation, microvascular dysfunction as well as the one of important reasons contributing to the tissue ischemia and cellular metabolic malfunction.

内皮细胞通透性升高是炎性反应的重要特点，是微血管功能障碍的主要表现，也是引起组织缺氧和细胞代谢障碍的重要原因之一。

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