Blood capillary of alveolar wall engorgement and pulmonary hyaline membrane are formed, and the infiltration of inflammatory cells in alveolar spaces. In the treatment group, these changes were less obvious, and the amplitude decreased.

  • 肺泡壁毛细血管充血扩张,有肺透明膜形成。肺间质、肺泡腔内有大量炎性细胞浸润,治疗组以上损伤程度明显减轻。
  • 来源:互联网摘选更新时间:2026-07-14 03:46:22

  • 重点词汇
  • theseadj. 这些(的)
  • theart.这个;指已提到或易领会到的人或事物;指独一无二的、正常的或不言而喻的人或事物;用以泛指;与形容词连用,指事物或统称的人;用于姓氏的复数形式前,指家庭或夫妇;(指特定用途的事物)足够,恰好;每,一;当前的,本,此;(重读,表示所指的为知名或重要的人或事物)
  • lessadv. 较少地,较小地;
  • changesn. (会令人感兴趣或可喜的)变化( change的名词复数 );换车;辅币;变迁;
  • treatmentn.治疗;对待;处理;
  • amplituden.振幅;广大,广阔,充足;(思想的)广度;角度距离;
  • were be的过去时复数和第二人称单数形式;有时代替 was,用于条件从句、动词 wish 之后等;
  • decreasedv.(使)缩短,减小,减少( decrease的过去式和过去分词 );
  • 相关例句
1、

Three months after Irradiation, acute inflammation performance disappear, a large number of fibroblast proliferation and the specific performance of mesenchymal cells increased, alveolar wall thickening, fibroblast cells increased, reducing the number of blood vessels.

照射3个月后急性炎症表现消失,出现成纤维细胞的大量增生,具体表现为间质细胞数增多、肺泡壁增厚、成纤维细胞数增多、血管数减少。

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2、

Microscope shows pulmono-stroma fibrous tissue hyperplasia, lymphocytes and neutrophil infiltrate, alveolar wall edema and thickening, alveolus cava contain with protein-material and a bit dust cells.

光镜下肺间质纤维组织增多、淋巴及中性粒细胞浸润,肺泡壁水肿、增厚,肺泡腔内含有蛋白样物质及少许尘埃细胞。

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3、

These studies demonstrated that, at least in animal models, apoptosis of alveolar wall or endothelial cells is sufficient to cause pulmonary emphysema, even without the accumulation of inflammatory cells.

动物实验证明即使没有炎性细胞的聚集,肺泡壁细胞或内皮细胞凋亡也足够引起肺气肿的发生。

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4、

The results showed epinephrine could increase the permeability of pulmonary microvascular wall and alveolar wall; there were edema, bleeding, transparent membranes in the tissue of damaged lung.

结果表明,肾上腺素可使肺血管壁、肺泡壁通透性增高,肺间质、肺泡、支气管水肿,出血及透明膜形成;

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5、

The pathological changes in lungs caused by PQ were mainly shown as congestion, leukocyte infiltration, thickness and local hemorrhage in alveolar wall.

肺组织病理学检查可见PQ中毒后肺泡壁充血、增厚,炎症细胞浸润,并有局灶性出血;

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6、

The important mechanical factors of lung injury might be tensile strain or stress in alveolar wall, and pulmonary superficial impact on the inside wall of lung.

肺损伤的重要力学因素可能是肺泡壁的拉伸应力或应变和肺表面的碰撞。

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7、

Ultrastructural characteristics of adventitial myofibroblast and cell phenotype in alveolar wall vessels were observed by electron microscopy.

透射电镜观察外膜成肌纤维细胞和腺泡内血管壁细胞表型。

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8、

Alveolar wall congestion, alveolar infiltration and gas bubbles in pulmonary capillary were observed in lung.

红细胞压积(Hct)升高。(2)快速减压可造成兔肺泡壁充血、肺泡腔内渗出,肺毛细血管内气泡形成。

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9、

Results: The congestion and bleeding of alveolar wall were lessened after given EDTA.

结果:给予EDTA后,肺泡壁充血、出血均有减轻,且高剂量组的各时期肺泡上皮细胞增生明显,易见核分裂。

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10、

Imaging Diagnosis and Classification of Alveolar Wall Vanishing Lung Syndrome with a Repot of 15 Cases

16例肺泡壁消失肺综合征影像学诊断及分型探讨

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11、

Group C alveolar wall thickening, between pulmonary edema, increased collagen deposition.

C组肺泡壁增厚,肺间质水肿,胶原纤维沉积增多。

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12、

Results: bmp3 was found in dental sac surrounding the germs at the early stage of tooth root development, and in cementoblasts and periodontal cells at the later stage.

结果:bmp3的表达始于牙根发育早期的牙囊组织,之后转移到成牙骨质细胞和牙周膜细胞中;

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13、

BMP may also be found in human tooth germs, predentin, the cells of outer and inner enamel epithelium, and the cells of dental sac generating alveolar bone.

在人的牙胚中,BMP主要分布在前期牙木质、外釉细胞和造釉细胞以及形成牙槽骨的牙囊细胞中,表明骨的生长和形成以及牙齿的发育都与BMR密切相关。

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14、

Result: During cap stage and early bell stage, the expression of MMP-20 mRNA was negative in enamel organ, dental papilla and dental sac and that of TIMP-1 mRNA could be observed in dental papilla;

结果:帽状期及钟状早期,成釉器、牙乳头及牙囊MMP-20mRNA表达阴性;TIMP-1mRNA在牙乳头表达;

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15、

Otherwise it was absent in normal dental sac or remnant dental lamina epithelia.

FGFR3在正常牙囊或残余牙板上皮中呈阴性表达。

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16、

Effect of different energy levels of pulsed Nd: YAG laser irradiation on the changes of SOD and MDA levels in ulcer tissue of oral ulcer model

脉冲Nd:YAG激光照射剂量对口腔溃疡组织中SOD活性和MDA含量的影响

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17、

Results: treatment group to ease the pain, oral ulcer, ulcer area and hyperemia area compared with the control group ( P 0.01).

结果:治疗组对缓解口腔溃疡的疼痛、缩小充血面积及溃疡面积与对照组比较(P0.01)。

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18、

Objective: To study the effects of soluble vascular endothelial cell adhesion molecule-1 ( sVCAM-1) and soluble intercellular adhesion molecule-1 ( sICAM-1) in recurrent oral ulceration ( ROU).

目的:探讨可溶性血管内皮细胞粘附分子(sVCAM1)、可溶性细胞间粘附分子(sICAM1)在复发性口腔溃疡(recurrent oral ulceration,ROU)发病机制中的作用。

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19、

Risk factors for CNS disease in HEV-71 included young age, fever, vomiting, mouth ulcers, breathlessness, cold limbs, and poor urine output.

易引起HEV-71感染者发生CNS疾病的危险因素包括年龄小、发热、呕吐、口腔溃疡、呼吸困难、四肢发冷和少尿。

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20、

Objective To investigate the association of recurrent aphthous ulcer ( RAU) with Helicobacter pylori ( Hp) infection and digestive diseases.

目的探讨复发性口腔溃疡(RAU)的发病与口腔幽门螺杆菌(Hp)感染及消化道疾病之间的关联。

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