I am sorry to keep you waiting?

Because there are too many orders, sorry to keep you waiting so long.

因为厨房订单太多，让您等了这么长时间。

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A ： Sorry to keep you waiting so long.

Sorry to keep you waiting so long.不好意思久等了。不好意思久等了。

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Sorry to keep you waiting. It's all right.

抱歉让你们久等了没关系

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Treatment effects of hypothermia on traumatic brain injury of sprague-dawley rat

局灶亚低温及全脑亚低温治疗大鼠创伤性脑损伤的实验研究

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It is revealed that erigeron breviscapus can relieve brain injury.

结论：灯盏花素治疗可以通过影响线粒体功能而减轻创伤性脑损伤后的继发性脑损害，从而改善其预后。

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DATA EXTRACTION: There were 65 literatures about the expression and role of S-100 β after cerebral ischemia, ischemia and anoxia encephalopathy, traumatic brain injury, central nervous system inflammation and epilepsy, and 28 with the representative viewpoints were cited.

资料提炼：检索到的关于脑缺血、创伤性脑损伤、缺血缺氧性脑病、中枢神经系统炎症和癫痫S-100β蛋白的表达与作用的文献共65篇，选择观点相似的文献引用28篇。

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Conclusions Early hyperbaric oxygenation could decrease the expression levels of AQP-4 and caspase-3, and ameliorate cytotoxic brain edema and decrease the apoptosis of neurocytes, which may provide the neuroprotective mechanism against brain traumas.

结论早期高压氧治疗能减少大鼠脑外伤后AQP-4和caspase-3的表达，高压氧对创伤性脑损伤的脑保护机制可能与减轻神经胶质细胞介导的细胞性脑水肿、减少神经细胞凋亡有关。

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Results Glutamate contents increased significantly at 15 minutes and reached a peak at 30 minutes, then decreased gradually at 6 hours and were inferior to normal level at 24 hours after brain injury;

结果创伤性脑损伤后15min神经细胞外液谷氨酸含量明显升高，伤后30min达高峰，伤后6h已开始降低，伤后24h降至最低值；

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Objective To investigate Changes of blood β-EP, ET in rats with traumatic brain injury following acute alcoholism ( TBIFAA) and its mechanism.

目的了解急性酒精中毒后创伤性脑损伤（TBIFAA）大鼠血β内啡呔（β-EP）、内皮素（ET）水平的改变，进一步探讨TBIFAA的创伤机制。

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Objective To study c-fos expression and apoptosis after traumatic brain injury ( TBI) in rats, and to explore the role of apoptosis in the secondary brain injury, and the relation between c-fos expression and neuroglial apoptosis.

目的研究创伤性脑损伤（TBI）后脑组织c-fos表达及神经细胞凋亡，探讨神经细胞凋亡在脑继发性损害中的作用，及c-fos基因表达与凋亡的关系。

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Conclusion The activation of NF-к Bp65 and ICAM-1 is related to brain inflammatory reaction, and which may be an important molecule mechanism of secondary brain injury after TBI.

结论创伤性脑损伤后NF-κBp65、ICAM-1的激活与脑炎性反应有关，是继发性脑损害的一个重要分子机制。

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AIM: To investigate the repairing effect of proliferation and transplantation of neural stem cells ( NSCs) on brain injury in rat model of traumatic brain injury ( TBI) according to the characteristics of self renewing and multi differentiation of NSCs.

目的：基于神经干细胞的自我更新和多向分化的特点，探讨创伤性脑损伤（traumatic brain injury，TBI）后神经干细胞（Neural Stem Cells，NSCs）的增殖及其移植对脑损伤的修复作用。

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The three indices BG_ ( 60-120), GIR_ ( 60-120), ISI reflecting the insulin sensitivity were measured 24 hours after severe traumatic brain injury by euglycemic-hyperinsulinemic clamp technique.

运用正常血糖-高血胰岛素钳夹技术，检测大鼠重型创伤性脑损伤后24h后BG60-120、GIR60-120、ISI等3个反映胰岛素敏感性的指标。

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Serum S-100 β protein level had close correlation with the severity and radiological findings after traumatic injury, and might be an index for poor prognosis.

创伤性脑损伤后S-100β血清水平的升高同损伤的严重程度和神经放射学发现均有相关性，并为创伤性脑损伤预后不良的指标。

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Objective To investigate the therapeutic effects of donepezil in the treatment of cognitive impairment and clinical prognosis after traumatic brain injury ( TBI).

目的探讨盐酸多奈哌齐对创伤性脑损伤（TBI）后认知功能障碍的干预作用及其对TBI临床预后的影响。

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Correlation of Brain Water Content with MRI Relaxation Time T_1 and Apparent Diffusion Coefficient Studied at 7.0 T in Two Models of Focal Cerebral Ischemia and Traumatic Brain Injury in the Rat

缺血与创伤性脑损伤T1、ADC动态变化及其与脑组织含水量相关性研究

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Objective To study the changes and effects of ceramide, interleukin 1 β( IL 1 β) and caspase 3 in cerebrum after traumatic brain injury ( TBI).

目的研究脑组织中白细胞介素-1β（IL-1β）、神经酰胺、caspase-3在创伤性脑损伤（TBI）后的改变和相互作用。

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The Expression of AQP-4 in Hippocampus of Rats after Moderate Traumatic Brain Injury Following Acute Alcoholism

急性酒精中毒合并中度创伤性脑损伤大鼠海马AQP4的表达

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The author summarized the status of recent studies on NF-κ B after TBI stressing on the mechanism of the changes and the double function of neuro-damage and cytoprotection.

笔者就近年来创伤性脑损伤后NFκB的研究进行如下综述，着重阐明脑损伤后NFκB活化的变化机制及其双重作用性。

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The Experimental and Clinical Studies on Apoptosis of Neural cells and its Relationship to Caspase-3 Gene Expression After Acute Traumatic Brain Injury

急性创伤性脑损伤后神经细胞凋亡及其与caspase-3基因表达关系的实验与临床研究

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