LPS

LPS Analysis of Chinese Strains of Coxiella Burnetii by SDS-PAGE and Immunoblot with Monoclonal Antibodies

我国Q热立克次体脂多糖的SDS-PAGE和单克隆抗体免疫印迹分析

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ACP, GOT, GPT, CHE, CK, LPS and LDH activities had little correlation with fiber digestibility and Met, Cys digestibility ( P & gt; 0.05 ).

酸性磷酸酶(ACP) 、 谷草转氨酶(GOT) 、 谷丙转氨酶(GPT) 、 胆碱酯酶(CHE) 、 肌酸激酶(CK) 、 脂肪激酶(LPS)和乳酸脱氢酶(LDH)的活性与营养物的消化率呈弱相关 ( P & gt; 0.05 ).

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Notably, treating mice with GSK-3 inhibitor showed a decrease in LPS-induced renal cell apoptosis in vivo.

除了抗发炎作用, GSK -3 抑制剂可以降低体内LPS诱发小鼠肾脏组织中的细胞凋亡现象.

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Conclusion LPS can stimulate expression of ESM 1 in endothelial cells.

结论　LPS对ECV304中ESM1的表达有促进作用.

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There were 76 ODPs and 285 LPs covering the HKSAR.

年底时,全港共有76份发展大纲图和285份发展蓝图.

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ConclusionThe soluble factors released by Kupffer cells, which were activated by LPS, could cause direct hepatocytes injury.

结论内毒素诱导库普弗细胞释放的可溶性因子作用一定时间后可直接对肝细胞造成损伤。

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These results indicate that LPS can increase the concentration of calcium in the elderly rats myocytes and has direct injury to the myocytes. 3.

上述结果表明，脂多糖通过造成心肌细胞内钙超负荷，对心肌细胞产生直接损伤作用。

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Objective: To observe the direct injury of lipopolysaccharide ( LPS) on cultured human umbilical vascular endothelial cells ( HUVEC) and the effect of IL 10 on it.

目的：观察脂多糖（LPS）对体外培养的人脐静脉内皮细胞（HUVEC）凋亡和坏死的直接作用以及IL10对其影响。

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Conclusion LPS could involve bone tissue in inflammation and inhibit the differentiation of osteoblast.

结论LPS刺激骨组织发生炎症反应、抑制成骨细胞分化.

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LPS could significantly increase mRNA expression of TNF-α and its secretion in dose-and time-dependent manners, which could be significantly attenuated by atorvastatin.

脂多糖能显著地升高TNF-α的mRNA表达及分泌水平，并呈时间和剂量依赖性，而阿托伐他汀能抑制该效应。

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In vitro experiments show that dark brick tea can inhibit LPS.

体外实验证明，青砖茶具有抑制LPS活性的作用。

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Lipopolysaccharide ( LPS) is a toxic component of cell walls of Gram-negative bacteria and is widely present in the gastrointestinal tracts of humans and animals.

脂多糖（lipopolysaccharide，LPS）是革兰氏阴性菌细胞壁组成成分，也是革兰氏阴性菌被人类和其他动物消化后释放的毒性成分，广泛存在于消化道中。

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Conclusion LPS can inhibit the apoptosis of PMNs.

结论LPS对PMNs的凋亡有抑制作用.

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Conclusion: The role of LPS in caries-induced reparative dentin might be a significant determinant.

结论: 提示适度的LPS在牙髓对龋病的防御反应过程中起着一定调节作用.

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At the end of 1998, there were 85 ODPs and 336 LPs.

迄一九九八年底, 已拟备的发展大纲图有85肺垃发展蓝图则有336份.

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There are evidences that several Gram-negative bacterial strains use their core LPS to interact with C-type lectin receptor.

由于近来发现的C型凝集素受体与某些革兰氏阴性菌的核心脂多糖（核心LPS）有作用。

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NF-κ B inhibitor CAPE suppressed LPS-induced these effects.

NFκB抑制剂咖啡酸苯乙酯(CAPE)抑制了LPS介导的上述效应.

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Subdiaphragmatic vagotomy reduce the responses of fever and c-Fos expression in rat PVN and NTS to LPS

膈下迷走神经切断降低LPS所致大鼠发热及室旁核、孤束核c-Fos蛋白表达

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In this reaction, PS was decomposed into polysilane of low molecu-lar weight ( LPS ) containing Si-C skeleton and Si-H bonds.

在这一反应中, PS裂解为含Si—H键的低分子聚硅烷 ( LPS ).

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CONCLUSION: LPS induces liver IL-6 expression , which might activate BEC proliferation through STAT 3 pathway.

结论: LPS刺激导致肝组织IL-6分泌增加, 后者可能通过STAT3介导BEC增殖.

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CD14 is a receptor of lipopolysaccharide ( LPS) of gram negative bacteria and expresses on membrane surface of phagocytes.

CD14作为革兰氏阴性菌细胞壁脂多糖的受体，表达在吞噬细胞膜表面上。

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PUPS can produce similar to LPS in murine peritoneal macrophages stimulated.

结论1.PUPS可以产生类似于LPS对小鼠腹腔巨噬细胞的刺激作用。

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Lipopolysaccharide ( LPS), a component of the outer membranes of Gram-negative bacteria, is a potent activator of macrophages and a major inducer of endotoxin shock.

脂多糖（1ipopolysaccharide，LPS）是革兰氏阴性菌外膜的主要成分，能够强烈激活巨噬细胞，是引起内毒素性休克的主要原因。

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In addition , apoptosis did occur in AEC isolated from TNF KO mice following LPS stimulation.

在TNF基因剔除(knockout,KO)小鼠,LPS仍 可明显诱导AEC凋亡.

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Results Histopathological evaluation: Typical pathological inflammation changes in the lung were observed in LPS groups, including alveolar congestion, hemorrhage, edema, infiltration of neutrophils in the airspace or vessel wall, thickness of the alveolar wall, and hyaline membrane formation.

【结果】肺组织病理学评价：LPS各组大鼠肺组织呈现典型的炎症病理变化，包括肺泡充血、出血、水肿、肺泡腔和血管壁中性粒细胞浸润、肺泡壁增厚、透明膜形成等病变。

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MAP , PaO _ 2 , pH and serum TNF-α concentrations were recor ded every hour after LPS.

5h内观察动物的MAP、PaO2 、 pH,血清TNF - α.

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The LPS concentration of plasma was detected by limulus lysate test. Application of Limulus Amoebocyte Lysate Reagent to Estimating Pyrogen in Antilymphocyte Immunoglobulin

采用鲎试剂法测血浆LPS浓度；鲎试剂在抗淋巴细胞免疫球蛋白热原检测中的应用

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The ultrastructure of Clara cells damaged significantly 24 h post LPS injection.

LPS致伤后24h,Clara细胞的超微结构明显受损.

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PurposeTo study extraction, purification and physicochemical analysis of Laminaria polysaccharide sulfate ( LPS ).

目的研究海带硫酸多糖 ( LPS ) 的提取制备及其组分的纯化和理化性质.

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The histologic changes of renal tissue were interstitial edema with inflammatory cellular infiltration, denaturalization and necrosis in some tubular epithelia. LPS caused 20% rats death.

肾间质水肿伴炎症细胞浸润，部分小管上皮细胞变性、坏死并见管型，死亡率20%。

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