LPs的例句大全

METHODS: The thiophosphoric acid modified oligonucleotides were transferred into rat peritoneal macrophages directly, TNF content in LPS-stimulated cell culture supernatant was determined by ELISA.

方法：直接将经硫代磷酸修饰的寡核苷酸导入大鼠腹腔巨噬细胞，以减少细胞分泌产生肿瘤坏死因子（TNF）的效果作为判断指标，以ELISA法检测细胞培养上清液中的TNF-α含量。

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Atorvastatin Reduces the LPS-induced COX-2 Expression in Cultured Human Pulmonary Epithelial Cells

阿托伐他汀降低脂多糖诱导下人肺上皮细胞COX-2的表达

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The Effect of Atorvastatin on TNF-α and IL-1 β in Peripheral Blood Monocytes Stimulated by LPS

阿托伐他汀对脂多糖刺激下外周血单核细胞分泌TNF-α、IL-1β的影响

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LPS could significantly increase mRNA expression of TNF-α and its secretion in dose-and time-dependent manners, which could be significantly attenuated by atorvastatin.

脂多糖能显著地升高TNF-α的mRNA表达及分泌水平，并呈时间和剂量依赖性，而阿托伐他汀能抑制该效应。

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Results Histopathological evaluation: Typical pathological inflammation changes in the lung were observed in LPS groups, including alveolar congestion, hemorrhage, edema, infiltration of neutrophils in the airspace or vessel wall, thickness of the alveolar wall, and hyaline membrane formation.

【结果】肺组织病理学评价：LPS各组大鼠肺组织呈现典型的炎症病理变化，包括肺泡充血、出血、水肿、肺泡腔和血管壁中性粒细胞浸润、肺泡壁增厚、透明膜形成等病变。

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The histologic changes of renal tissue were interstitial edema with inflammatory cellular infiltration, denaturalization and necrosis in some tubular epithelia. LPS caused 20% rats death.

肾间质水肿伴炎症细胞浸润，部分小管上皮细胞变性、坏死并见管型，死亡率20%。

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The LPS concentration of plasma was detected by limulus lysate test. Application of Limulus Amoebocyte Lysate Reagent to Estimating Pyrogen in Antilymphocyte Immunoglobulin

采用鲎试剂法测血浆LPS浓度；鲎试剂在抗淋巴细胞免疫球蛋白热原检测中的应用

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a study of the structure of molecular chain and aggregation state, thermal performance and dilute solution property of ring shaped ps ( rps) was made by several analyses and determinations, and a comparison was also made with linear shaped ps ( lps).

本文通过多种分析测试手段，详细研究了环型聚苯乙烯（RPS）分子链结构、聚集态结构、热性能及稀溶液性质，并与线型聚苯乙烯（LPS）作了对照。

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the change of cytokines and nuclear factor-kappa b in lung tissue of lps-induced acute lung injury in neonatal rats: the effect of n-acetylcysteine

LPS致新生大鼠急性肺损伤促炎性细胞因子、抗炎性细胞因子和NF-κB变化及N-乙酰半胱氨酸干预作用的研究

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bp-lps slightly increased the level of serum hemolysin, intensity of reaction of dth, macrophage phagocytosis and peripheral white blood cell count in normal mice, but there was no significant difference vs control group. 4.

BP-LPS对正常小鼠血清溶血素水平、迟发型超敏反应、单核吞噬细胞吞噬功能以及外周血白细胞数有轻微促进作用，但是与对照组比较无统计学意义。

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objective: to study the effects of pentoxifylline ( ptx) on pulmonary microvascular permeability of lps induced acute lung injury.

目的：探讨己酮可可碱（PTX）对内毒素（L PS）诱导的急性肺损伤肺微血管通透性的影响。

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objective to investigate the effects of low-dose hydrocortisone on brain dysfunction in lps-induced sepsis rats and the role of nuclear factor kappa b ( nf-κ b) signal transcription pathway in the pathogenesis.

目的研究小剂量氢化可的松（HC）对内毒素诱导的脓毒症大鼠脑功能障碍的影响及核因子κB（NFκB）信号转导途径在发病机制中的作用。

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PUPS can produce similar to LPS in murine peritoneal macrophages stimulated.

结论1.PUPS可以产生类似于LPS对小鼠腹腔巨噬细胞的刺激作用。

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These results indicate that LPS can increase the concentration of calcium in the elderly rats myocytes and has direct injury to the myocytes. 3.

上述结果表明，脂多糖通过造成心肌细胞内钙超负荷，对心肌细胞产生直接损伤作用。

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Objective: To observe the direct injury of lipopolysaccharide ( LPS) on cultured human umbilical vascular endothelial cells ( HUVEC) and the effect of IL 10 on it.

目的：观察脂多糖（LPS）对体外培养的人脐静脉内皮细胞（HUVEC）凋亡和坏死的直接作用以及IL10对其影响。

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ConclusionThe soluble factors released by Kupffer cells, which were activated by LPS, could cause direct hepatocytes injury.

结论内毒素诱导库普弗细胞释放的可溶性因子作用一定时间后可直接对肝细胞造成损伤。

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In vitro experiments show that dark brick tea can inhibit LPS.

体外实验证明，青砖茶具有抑制LPS活性的作用。

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Subdiaphragmatic vagotomy reduce the responses of fever and c-Fos expression in rat PVN and NTS to LPS

膈下迷走神经切断降低LPS所致大鼠发热及室旁核、孤束核c-Fos蛋白表达

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Lipopolysaccharide ( LPS) is one of the most important causative agents which induced ALI and the mechanism is still unclear.

脂多糖（Lipopolysaccharide，LPS）是严重感染所致ALI的最重要诱发原因之一，其致病机理依然不明，探讨LPS导致ALI的机理是这一领域的研究热点。

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Influence of the serum of Radix Ophiopogonis on the apoptosis related gene expression and intracellular Ca~ ( 2+) in vascular endothelial cells treated with LPS

麦冬药物血清对血管内皮细胞凋亡相关基因表达及胞内Ca~（2+）的影响

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